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341272

Sigma-Aldrich

Anti-Factor B Goat pAb

liquid, Calbiochem®

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About This Item

Código UNSPSC:
12352203
NACRES:
NA.43

origen biológico

goat

Nivel de calidad

forma del anticuerpo

serum

tipo de anticuerpo

primary antibodies

clon

polyclonal

Formulario

liquid

contiene

≤0.1% sodium azide as preservative

reactividad de especies

human

fabricante / nombre comercial

Calbiochem®

disponibilidad

not available in Canada

condiciones de almacenamiento

OK to freeze
avoid repeated freeze/thaw cycles

isotipo

IgG

Condiciones de envío

ambient

temp. de almacenamiento

−20°C

modificación del objetivo postraduccional

unmodified

Información sobre el gen

human ... CFB(629)

Descripción general

Goat polyclonal antibody supplied as undiluted serum. Recognizes Factor B.
Recognizes Factor B.
This Anti-Factor B Goat pAb is validated for use in Immunoelectrophoresis, Radial Immunodiffusion, Ouchterlony for the detection of Factor B.

Inmunógeno

Human
purified, human factor B

Aplicación

Immunoelectrophoresis (see comments)

Radial Immunodiffusion (see comments)

Ouchterlony (≥ undiluted)

Envase

Please refer to vial label for lot-specific concentration.

Advertencia

Toxicity: Standard Handling (A)

Reconstitución

Following initial thaw, aliquot and freeze (-20°C).

Otras notas

This antibody has also been reported to work for immunoelectrophoresis and radial immunodiffusion. Variables associated with assay conditions will dictate the proper working dilution.

Información legal

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

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Código de clase de almacenamiento

10 - Combustible liquids

Clase de riesgo para el agua (WGK)

WGK 1


Certificados de análisis (COA)

Busque Certificados de análisis (COA) introduciendo el número de lote del producto. Los números de lote se encuentran en la etiqueta del producto después de las palabras «Lot» o «Batch»

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C Duehrkop et al.
Laboratory animals, 48(2), 143-154 (2014-03-04)
Prolonged ischemia of skeletal muscle tissue, followed by reperfusion, leads to ischemia/reperfusion injury (IRI), which is a feared local and systemic inflammatory reaction. With respect to the 3Rs, we wanted to determine which parameters for assessment of IRI require a
Diana Pauly et al.
Cell reports, 29(9), 2835-2848 (2019-11-28)
Complement dysregulation is a feature of many retinal diseases, yet mechanistic understanding at the cellular level is limited. Given this knowledge gap about which retinal cells express complement, we performed single-cell RNA sequencing on ∼92,000 mouse retinal cells and validated
Anna M Blom et al.
Journal of clinical immunology, 36(5), 517-527 (2016-05-06)
Autoantibodies termed C3-nephritic factor (C3NeF), which stabilize convertases of the alternative complement pathway, often stimulate autoinflammatory diseases. However, knowledge about analogous autoantibodies acting on the classical pathway (C4NeF) is limited to a few reports, which indicate association with kidney dysfunction
Claudia Duehrkop et al.
PloS one, 8(8), e72059-e72059 (2013-08-31)
Ischemia/reperfusion injury of lower extremities and associated lung damage may result from thrombotic occlusion, embolism, trauma, or surgical intervention with prolonged ischemia and subsequent restoration of blood flow. This clinical entity is characterized by high morbidity and mortality. Deprivation of
Filippo Mori et al.
Frontiers in immunology, 15, 1334151-1334151 (2024-06-26)
Complement factor H (FH) is a major regulator of the complement alternative pathway, its mutations predispose to an uncontrolled activation in the kidney and on blood cells and to secondary C3 deficiency. Plasma exchange has been used to correct for

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