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449172

Sigma-Aldrich

Magnesium chloride

AnhydroBeads, −10 mesh, 99.99% trace metals basis

Synonym(s):

Magnogene

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About This Item

Linear Formula:
MgCl2
CAS Number:
Molecular Weight:
95.21
EC Number:
MDL number:
UNSPSC Code:
12352302
PubChem Substance ID:
NACRES:
NA.23

product line

AnhydroBeads

Quality Level

Assay

99.99% trace metals basis

impurities

≤150.0 ppm Trace Metal Analysis

particle size

−10 mesh

mp

714 °C (lit.)

density

2.32 g/mL at 25 °C (lit.)

SMILES string

Cl[Mg]Cl

InChI

1S/2ClH.Mg/h2*1H;/q;;+2/p-2

InChI key

TWRXJAOTZQYOKJ-UHFFFAOYSA-L

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General description

Magnesium chloride can be used as a starting material to prepare electrolyte solutions for Mg-polysulfide flow batteries.

Legal Information

AnhydroBeads is a trademark of Sigma-Aldrich Co. LLC

Storage Class Code

13 - Non Combustible Solids

WGK

WGK 1

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Role of chloride for a simple, non-grignard Mg electrolyte in ether-based solvents
Niya Sa, et al
ACS Applied Materials & Interfaces, 8, 16002-16008 (2016)
Stability and Disproportionation of Magnesium Polysulfides and the Effects on the Mg-Polysulfide Flow Battery
Peng He, et al.
Journal of the Electrochemical Society, 168, 110516-110516 (2021)
Manal A Farg et al.
Human molecular genetics, 23(13), 3579-3595 (2014-02-20)
Intronic expansion of a hexanucleotide GGGGCC repeat in the chromosome 9 open reading frame 72 (C9ORF72) gene is the major cause of familial amyotrophic lateral sclerosis (ALS) and frontotemporal dementia. However, the cellular function of the C9ORF72 protein remains unknown.
Fernando Cartón-García et al.
Scientific reports, 5, 12312-12312 (2015-07-24)
Inherited MYO5B mutations have recently been associated with microvillus inclusion disease (MVID), an autosomal recessive syndrome characterized by intractable, life-threatening, watery diarrhea appearing shortly after birth. Characterization of the molecular mechanisms underlying this disease and development of novel therapeutic approaches
Benjamin Dombert et al.
PloS one, 9(10), e110846-e110846 (2014-10-23)
Spinal muscular atrophy (SMA) is caused by deficiency of the ubiquitously expressed survival motoneuron (SMN) protein. SMN is crucial component of a complex for the assembly of spliceosomal small nuclear ribonucleoprotein (snRNP) particles. Other cellular functions of SMN are less

Protocols

Summary application report for analysis of moisture in Magnesium chloride

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