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Key Documents

292265

Sigma-Aldrich

N,N′-Diphenyl-p-phenylenediamine

98%

Synonym(s):

N,N′-Diphenyl-1,4-phenylenediamine

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About This Item

Linear Formula:
C6H5NHC6H4NHC6H5
CAS Number:
Molecular Weight:
260.33
Beilstein:
2215944
EC Number:
MDL number:
UNSPSC Code:
12162002
PubChem Substance ID:
NACRES:
NA.23

Quality Level

Assay

98%

bp

220-225 °C/0.5 mmHg (lit.)

mp

143-145 °C (lit.)

SMILES string

N(c1ccccc1)c2ccc(Nc3ccccc3)cc2

InChI

1S/C18H16N2/c1-3-7-15(8-4-1)19-17-11-13-18(14-12-17)20-16-9-5-2-6-10-16/h1-14,19-20H

InChI key

UTGQNNCQYDRXCH-UHFFFAOYSA-N

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Application

  • TPA-induced inhibition of gap junctional intercellular communication is not mediated through free radicals.: This research explores the role of N,N′-Diphenyl-p-phenylenediamine (DPPD) in cellular communication, providing insights into its applications in high-performance rubber stabilizers and life science manufacturing chemicals. (Hasler et al., 1990).

Pictograms

Exclamation mark

Signal Word

Warning

Hazard Statements

Hazard Classifications

Aquatic Chronic 3 - Skin Sens. 1

Storage Class Code

11 - Combustible Solids

WGK

WGK 1

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Yoshiko Kawai et al.
Journal of pharmacological sciences, 111(4), 433-439 (2009-12-08)
Cisplatin causes chronic interstitial disease with fibrosis, but the development mechanism of interstitial fibrosis is not yet understood. We examined the effect of an antioxidant, N,N'-diphenyl-1,4-phenylenediamine (DPPD), on development of interstitial fibrosis induced by cisplatin. Cisplatin increased blood urea nitrogen
Sung Min Park et al.
Pharmacology & toxicology, 92(1), 43-50 (2003-04-25)
This study was undertaken in order to examine the roles of lipid peroxidation and poly (ADP-ribose) polymerase (PARP) activation in oxidant-induced renal cell death. Opossum kidney cell cultures were used as the renal epithelial cell model, and an inorganic hydroperoxide
I Rath et al.
Zeitschrift fur Naturforschung. C, Journal of biosciences, 55(11-12), 957-964 (2001-02-24)
The primary reactions leading to Al toxicity in plant cells have not yet been elucidated. We used soybean (Glycine max [L.] Merr.) cell suspension cultures to address the question whether lipid peroxidation plays an important role in Al toxicity. Upon
A Campos-Neto et al.
Tuberculosis (Edinburgh, Scotland), 81(5-6), 353-358 (2002-01-22)
Although the tuberculin test has aided in the diagnosis of tuberculosis for more than 85 years, its interpretation is difficult particularly because sensitization with non-tuberculous mycobacteria leads to false positive tests. Using the guinea pig model of tuberculosis, we have
Arun Radhakrishnan
Biophysical journal, 98(9), L41-L43 (2010-05-06)
A pair of recent studies has reopened debate on the subject of phase separations in model bilayer mixtures of cholesterol (Chol) and dipalmitoyl-phosphatidylcholine (DPPC). Fluorescence microscopy methods have not been able to detect phase separations in binary DPPC-Chol mixtures that

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