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一般說明
SigmaScreen 链霉亲和素包被的高容量微孔板采用了专有的包被技术，与标准链霉亲和素或 ExtrAvidin 包被微孔板相比，生物素结合能力大大增强。这种透明的 96 孔板在单孔托架上具有易碎的 8 孔条的形式，并且具有≥ 15/孔的结合能力。链霉亲和素包被的高容量微孔板提供了一个平台，可洗脱捕获的蛋白，通过 MALDI、ICAT 或 SDS-PAGE 等多种方法进行捕获后分析。
法律資訊
SigmaScreen is a trademark of Sigma-Aldrich Co. LLC
儲存類別代碼
11 - Combustible Solids
水污染物質分類（WGK）
WGK 2
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Gloves, type N95 (US)
Lu Li et al.
Analytica chimica acta, 886, 123-132 (2015-09-01)
A method for fabrication of multiplexed optical coding nanobeads (MOCNBs) was developed by hybridizing three types of coding DNAs labeled with different dyes (Cy5, FAM and AMCA) at precisely controlled ratios with biotinylated reporter DNA modified to magnetic streptavidin-coated nanobeads
Hong-Sheng Lim et al.
Journal of immunology (Baltimore, Md. : 1950), 195(11), 5432-5439 (2015-10-27)
Optimal T cell activation typically requires engagement of both the TCR and costimulatory receptors, such as CD28. Engagement of CD28 leads to tyrosine phosphorylation of its cytoplasmic region and recruitment of cytoplasmic signaling proteins. Although the exact mechanism of CD28
Román González-Prieto et al.
Cell reports, 34(4), 108691-108691 (2021-01-28)
In contrast to our extensive knowledge on covalent small ubiquitin-like modifier (SUMO) target proteins, we are limited in our understanding of non-covalent SUMO-binding proteins. We identify interactors of different SUMO isoforms-monomeric SUMO1, monomeric SUMO2, or linear trimeric SUMO2 chains-using a
Yasmin ElTahir et al.
BMC molecular and cell biology, 20(1), 55-55 (2019-12-01)
Brucella is a facultative intracellular pathogen responsible for zoonotic disease brucellosis. Little is known about the molecular basis of Brucella adherence to host cells. In the present study, the possible role of Bp26 protein as an adhesin was explored. The
Jie Wang et al.
Nature, 569(7757), 509-513 (2019-05-10)
A universal gain-of-function approach for selective and temporal control of protein activity in living systems is crucial to understanding dynamic cellular processes. Here we report development of a computationally aided and genetically encoded proximal decaging (hereafter, CAGE-prox) strategy that enables time-resolved
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Protein modifications are crucial for disease study. Analysis methods are key.
Protein modifications are crucial for disease study. Analysis methods are key.
Protein modifications are crucial for disease study. Analysis methods are key.
Protein modifications are crucial for disease study. Analysis methods are key.
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