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Merck

78533

Supelco

for TOC analysis

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About This Item

线性分子式:
H2O
CAS号:
分子量:
18.02
Beilstein:
2050024
EC號碼:
MDL號碼:
分類程式碼代碼:
12191602
PubChem物質ID:
NACRES:
NA.21

蒸汽密度

<1 (vs air)

品質等級

蒸汽壓力

3 mmHg

儲存期限

limited shelf life, expiry date on the label

雜質

≤0.10 mg/L TOC

折射率

n20/D 1.34 (lit.)

bp

100 °C (lit.)

mp

0 °C (lit.)

密度

1.000 g/mL at 3.98 °C (lit.)

適合性

suitable for (TOC analysis)

應用

environmental
food and beverages
general analytical

SMILES 字串

O

InChI

1S/H2O/h1H2

InChI 密鑰

XLYOFNOQVPJJNP-UHFFFAOYSA-N

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一般說明

总有机碳分析是一种测量水中存在的溶解和颗粒有机物碳含量的方法。TOC 分析有助于去除无机碳,将有机碳氧化为 CO 2 并定量 CO 2

儲存類別代碼

12 - Non Combustible Liquids

水污染物質分類(WGK)

nwg

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

Eyeshields, Gloves


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分析证书(COA)

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Frank R. Spellman
Handbook of Water and Wastewater Treatment Plant Operations, 50-50 (2008)
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Oseltamivir-resistant H1N1 influenza viruses carrying the H275Y neuraminidase mutation predominated worldwide during the 2007-2009 seasons. Although several neuraminidase substitutions were found to be necessary to counteract the adverse effects of H275Y, the order and impact of evolutionary events involved remain
Mary A Rodgers et al.
The Journal of experimental medicine, 211(7), 1333-1347 (2014-06-25)
Linear ubiquitination is a newly discovered posttranslational modification that is currently restricted to a small number of known protein substrates. The linear ubiquitination assembly complex (LUBAC), consisting of HOIL-1L, HOIP, and Sharpin, has been reported to activate NF-κB-mediated transcription in
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PLoS pathogens, 10(6), e1004192-e1004192 (2014-06-20)
Highly pathogenic avian influenza (HPAI) viruses of the H5N1 subtype often cause severe pneumonia and multiple organ failure in humans, with reported case fatality rates of more than 60%. To develop a clinical antibody therapy, we generated a human-mouse chimeric
M Grusdat et al.
Cell death and differentiation, 21(7), 1050-1060 (2014-02-18)
CD8(+) T-cell functions are critical for preventing chronic viral infections by eliminating infected cells. For healthy immune responses, beneficial destruction of infected cells must be balanced against immunopathology resulting from collateral damage to tissues. These processes are regulated by factors

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