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AB5716

Sigma-Aldrich

Anti-Hey 2/HRT2 Antibody

Chemicon®, from rabbit

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

rabbit

Quality Level

100

antibody form

affinity purified immunoglobulin

antibody product type

primary antibodies

clone

polyclonal

purified by

affinity chromatography

species reactivity

human, mouse

manufacturer/tradename

Chemicon®

technique(s)

immunohistochemistry: suitable

NCBI accession no.

NM_012259.2

UniProt accession no.

Q9UBP5

shipped in

dry ice

target post-translational modification

unmodified

Gene Information

human ... HEY2(23493)

Specificity

Recognizes Hey 2 / HRT2, a Helix-loop-helix class of transcription factor.

Immunogen

Synthetic peptide, amino acids 314-330 of human Hey 2 / HRT2.

Application

Anti-Hey 2/HRT2 Antibody is an antibody against Hey 2/HRT2 for use in IH.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Sameer H Halani et al.
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Oligodendrogliomas are diffusely infiltrative gliomas defined by IDH-mutation and co-deletion of 1p/19q. They have highly variable clinical courses, with survivals ranging from 6 months to over 20 years, but little is known regarding the pathways involved with their progression or
Expression of HES and HEY genes in infantile hemangiomas.
Adepoju, O; Wong, A; Kitajewski, A; Tong, K; Boscolo, E; Bischoff, J; Kitajewski, J; Wu, JK
Vascular Cell null
Madhumita Basu et al.
JCI insight, 2(20) (2017-10-20)
Birth defects are the leading cause of infant mortality, and they are caused by a combination of genetic and environmental factors. Environmental risk factors may contribute to birth defects in genetically susceptible infants by altering critical molecular pathways during embryogenesis
Danielle Daley-Brown et al.
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The expression of NILCO molecules (Notch, IL-1, and leptin crosstalk outcome) and the association with obesity were investigated in types I and II endometrial cancer (EmCa). Additionally, the involvement of NILCO in leptin-induced invasiveness of EmCa cells was investigated. The
Ana-Rita Pedrosa et al.
The Prostate, 76(1), 80-96 (2015-10-01)
The Notch signaling pathway has been implicated in prostate development, maintenance and tumorigenesis by its key role in cell-fate determination, differentiation and proliferation. Therefore, we proposed to analyze Notch family members transcription and expression, including ligands (Dll1, 3, 4 and
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