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343226

Sigma-Aldrich

Nickel sulfide

99.7% trace metals basis, −150 mesh

Synonym(s):

Nickel subsulfide

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About This Item

Empirical Formula (Hill Notation):
Ni3S2
CAS Number:
12035-72-2
Molecular Weight:
240.21
EC Number:
234-829-6
MDL number:
MFCD00217044
UNSPSC Code:
12161600
PubChem Substance ID:
24861133
NACRES:
NA.23

Assay

99.7% trace metals basis

form

powder

reaction suitability

reagent type: catalyst
core: nickel

particle size

−150 mesh

density

5.8 g/mL at 25 °C (lit.)

application(s)

battery manufacturing

SMILES string

[Ni]=S=[Ni]=S=[Ni]

InChI

1S/3Ni.2S

InChI key

YGHCWPXPAHSSNA-UHFFFAOYSA-N

General description

Major phase is Ni3S2. Product may also include a small amount of sulfur-rich phase. Nickel Sulfide (Ni3S2) exists as a heazlewoodite mineral in nature. The structure of Ni3S2 is hombohedral. Raman spectral analysis of Ni3S2 has been reported.

Application

Nickel sulfide was implanted in mice, to induce the growth of malignant fibrous histiocytomas.

Packaging

Packaged in glass bottles

Signal Word

Danger

Hazard Classifications

Acute Tox. 3 Inhalation - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A Inhalation - Muta. 2 - Skin Sens. 1 - STOT RE 1 Inhalation

Target Organs

Lungs

Storage Class Code

6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Aaron Barchowsky et al.
The Journal of biological chemistry, 277(27), 24225-24231 (2002-04-30)
Inhalation of particulate nickel subsulfide (Ni3S2) causes chronic active inflammation and fibrosis of the lungs. However, the mechanisms for these effects are not well understood. Therefore, cell culture experiments with BEAS-2B human airway epithelial cells were conducted to test the
Joseph R Landolph et al.
Environmental health perspectives, 110 Suppl 5, 845-850 (2002-11-12)
In the past, exposure of workers to mixtures of soluble and insoluble nickel compounds by inhalation during nickel refining correlated with increased incidences of lung and nasal cancers. Insoluble nickel subsulfide and nickel oxide (NiO) are carcinogenic in animals by
M Okamoto
Differentiation; research in biological diversity, 61(5), 285-292 (1997-08-01)
It is well known that urodeles have the most powerful regenerative capacities among vertebrates, but there is little realisation that they are extremely resistant to spontaneous or chemically induced tumours. Regeneration and carcinogenesis have been considered to be two sides
X Mao et al.
Cancer letters, 161(1), 57-62 (2000-11-18)
Differential display polymerase chain reaction (DD-PCR) was used to analyze the differentially expressed genes from nickel-transformed human embryonic lung (HEL) cells (MRC-9 and IMR-90) and their control counterparts (non-treated). Two genes, MS515 and IC82, were confirmed by Northern blot analysis.
C Huang et al.
Cancer research, 61(22), 8051-8057 (2001-11-24)
Nickel compounds induce cell transformation in cell culture models and tumor formation in experimental animals. However, the molecular mechanisms by which nickel compounds induce tumors are not yet well understood. The present study found that exposure of cells to either
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