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P7658

Sigma-Aldrich

Poly(D-Glu, D-Lys) hydrobromide

suitable for ligand binding assays, Mol wt 20,000-50,000

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About This Item

CAS Number:
MDL number:
UNSPSC Code:
12352202
NACRES:
NA.26

product name

Poly(D-Glu, D-Lys) hydrobromide, D-Glu:D-Lys (6:4), mol wt 20,000-50,000

form

powder

Quality Level

feed ratio

D-Glu:D-Lys (6:4)

mol wt

20,000-50,000

technique(s)

ligand binding assay: suitable

color

white

storage temp.

−20°C

Analysis Note

Molecular weight based on viscosity.

Other Notes

For additional technical information on polyamino acids please visit the Polyamino acid FAQ resource.

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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F Borek et al.
The Biochemical journal, 96(3), 577-582 (1965-09-01)
1. Three random linear copolymers composed of two or three of the amino acids d-tyrosine, d-glutamic acid, d-alanine and d-lysine, and a branched multichain copolymer with a poly-d-lysine backbone and polymeric side chains of d-tyrosine and d-glutamic acid, were found
F T Liu et al.
Proceedings of the National Academy of Sciences of the United States of America, 76(3), 1430-1434 (1979-03-01)
Administration of stable conjugates prepared by coupling protein antigens such as ovalbumin or antigen E of ragweed extract to the synthetic random copolymer of D-glutamic acid and D-lysine (D-GL) is effective in inducing a state of long-lasting, antigen-specific immunological tolerance
P S Norman et al.
The Journal of allergy and clinical immunology, 73(6), 782-789 (1984-06-01)
Allergens conjugated with several simple repeating polymers have reduced allergenicity in man, but large doses retain the ability to suppress ongoing allergen-specific IgE synthesis in strains of high-responder mice. To determine whether suppression of IgE antibodies could be induced in

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