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Merck
모든 사진(1)

주요 문서

SML2746

Sigma-Aldrich

SRS11-92

≥98% (HPLC)

동의어(들):

4-(Cyclohexylamino)-3-[(phenylmethyl)amino]-benzoic acid ethyl ester, Ethyl 3-(benzylamino)-4-(cyclohexylamino)benzoate

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About This Item

실험식(Hill 표기법):
C22H28N2O2
CAS Number:
Molecular Weight:
352.47
UNSPSC 코드:
12352200
NACRES:
NA.77

Quality Level

분석

≥98% (HPLC)

양식

powder

색상

white to yellow-brown

solubility

DMSO: 2 mg/mL, clear

저장 온도

2-8°C

SMILES string

N(C3CCCCC3)c1c(cc(cc1)C(=O)OCC)NCc2ccccc2

InChI key

VHQAJFNLPQULSV-UHFFFAOYSA-N

생화학적/생리학적 작용

SRS11-92 is a cell penetrant and potent inhibitor of ferroptosis that attenuates the cell death associated with frataxin knockdown in healthy human fibroblasts. SRS11-92 protects against cell death in in a brain slice model of Huntington′s disease, an oligodendrocyte model of periventricular leukomalacia, and in isolated kidney proximal tubules model of kidney disfunction.
cell penetrant and potent inhibitor of ferroptosis that attenuates the cell death associated with frataxin knockdown in healthy human fibroblasts

Storage Class Code

11 - Combustible Solids

WGK

WGK 3


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문서 라이브러리 방문

Sam Hofmans et al.
Journal of medicinal chemistry, 59(5), 2041-2053 (2015-12-24)
Ferroptosis is a nonapoptotic, iron-catalyzed form of regulated necrosis that is critically dependent on glutathione peroxidase 4 (GPX4). It has been shown to contribute to liver and kidney ischemia reperfusion injury in mice. A chemical inhibitor discovered by high-throughput screening
Rachid Skouta et al.
Journal of the American Chemical Society, 136(12), 4551-4556 (2014-03-07)
Ferrostatin-1 (Fer-1) inhibits ferroptosis, a form of regulated, oxidative, nonapoptotic cell death. We found that Fer-1 inhibited cell death in cellular models of Huntington's disease (HD), periventricular leukomalacia (PVL), and kidney dysfunction; Fer-1 inhibited lipid peroxidation, but not mitochondrial reactive
M Grazia Cotticelli et al.
The Journal of pharmacology and experimental therapeutics, 369(1), 47-54 (2019-01-13)
Friedreich ataxia (FRDA) is a progressive neuro- and cardio-degenerative disorder characterized by ataxia, sensory loss, and hypertrophic cardiomyopathy. In most cases, the disorder is caused by GAA repeat expansions in the first introns of both alleles of the FXN gene

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