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Merck
모든 사진(1)

주요 문서

EMU090361

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Timp2

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About This Item

UNSPSC 코드:
41105324
NACRES:
NA.51

설명

Powered by Eupheria Biotech

Quality Level

제품 라인

MISSION®

양식

lyophilized powder

esiRNA cDNA 표적 서열

ATCTATGGCAACCCCATCAAGAGGATTCAGTATGAGATCAAGCAGATAAAGATGTTCAAAGGACCTGACAAAGACATCGAGTTTATCTACACGGCCCCCTCTTCAGCAGTGTGCGGGGTCTCGCTGGACGTTGGAGGAAAGAAGGAGTATCTAATTGCAGGAAAGGCAGAAGGAGATGGCAAGATGCACATTACCCTCTGTGACTTCATTGTGCCCTGGGACACGCTTAGCATCACCCAGAAGAAGAGCCTGAACCACAGGTACCAGATGGGCTGTGAGTGCAAGATCACTCGCTGTCCCATGATCCCTTGCTACATCTCCTCCCCGGATGAGTGCCTCTGGATGGACTGGGTCACAGAGAAGAGCATCAATGGGCACCAGGCCAAGTTCTTCGCCTGCATCAAGAGAAGTGATGGTTCTTGCGCGTGGTACCGCGGGGCGGCACCCCCCAAGCAAGAGTTTCTTGACATCGAGGACCCGTAAGAAGGCTGACAGAGCCCCTGTGGCCAATTGAAAAGCCTCTGAGGGTTTAGACTGGTCCAGCTTTGACATCCCTTCCTGGA

Ensembl | 마우스 수납 번호

NCBI 수납 번호

배송 상태

ambient

저장 온도

−20°C

유전자 정보

일반 설명

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

법적 정보

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class Code

10 - Combustible liquids

Flash Point (°F)

Not applicable

Flash Point (°C)

Not applicable


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시험 성적서(COA)

Lot/Batch Number

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문서 라이브러리 방문

An Yan et al.
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 37(1), 55-66 (2015-08-19)
The aim of this study was to investigate the role of microRNA miR-761 in the progression and metastasis of non-small cell lung cancer (NSCLC), and the mechanisms by which miR-761 regulates cell proliferation and metastatic activity of NSCLC cell lines.
Qinhong Xu et al.
Oncotarget, 6(16), 14153-14164 (2015-04-18)
MicroRNAs are involved in the initiation and progression of pancreatic cancer. In this study, we showed that miR-221/222 is overexpressed in pancreatic cancer. MiR-221/222 overexpression significantly promoted pancreatic cancer cell proliferation and invasion while inhibiting apoptosis. The expression of the

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