アッセイ
99.95%
形状
foil
メーカー／製品名
Goodfellow 808-025-69
抵抗性
1.673 μΩ-cm, 20°C
サイズ×厚み
100 x 100 mm × 0.010 mm
bp
2567 °C (lit.)
mp
1083.4 °C (lit.)
密度
8.94 g/mL at 25 °C (lit.)
SMILES記法
[Cu]
InChI
1S/Cu
InChI Key
RYGMFSIKBFXOCR-UHFFFAOYSA-N
詳細
For updated SDS information please visit www.goodfellow.com.
法的情報
Goodfellow製品
試験成績書（COA）
製品のロット番号・バッチ番号を入力して、試験成績書（COA） を検索できます。ロット番号・バッチ番号は、製品ラベルに「Lot」または「Batch」に続いて記載されています。
Julie E Gleason et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(16), 5866-5871 (2014-04-09)
The human fungal pathogens Candida albicans and Histoplasma capsulatum have been reported to protect against the oxidative burst of host innate immune cells using a family of extracellular proteins with similarity to Cu/Zn superoxide dismutase 1 (SOD1). We report here
Adam Southon et al.
Metallomics : integrated biometal science, 5(10), 1346-1356 (2013-08-02)
Copper (Cu) is an essential redox active metal that is potentially toxic in excess. Multicellular organisms acquire Cu from the diet and must regulate uptake, storage, distribution and export of Cu at both the cellular and organismal levels. Systemic Cu
Giulia Cheloni et al.
Aquatic toxicology (Amsterdam, Netherlands), 155, 275-282 (2014-07-30)
The present study showed the important role of light intensity and spectral composition on Cu uptake and effects on green alga Chlamydomonas reinhardtii. High-intenisty light (HL) increased cellular Cu concentrations, but mitigated the Cu-induced decrease in chlorophyll fluorescence, oxidative stress
Alina Fedoseienko et al.
Annals of the New York Academy of Sciences, 1314, 6-14 (2014-04-05)
Copper is an important cofactor in numerous biological processes in all living organisms. However, excessive copper can be extremely toxic, so it is vital that the copper level within a cell is tightly regulated. The damaging effect of copper is
Lelita T Braiterman et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
ライフサイエンス、有機合成、材料科学、クロマトグラフィー、分析など、あらゆる分野の研究に経験のあるメンバーがおります。.
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