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Merck

GF31833413

foil, 6mm disks, thickness 0.008mm, 99.85%

別名:

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About This Item

化学式:
Fe
CAS番号:
分子量:
55.85
MDL番号:
UNSPSCコード:
12141721
PubChem Substance ID:
NACRES:
NA.23

アッセイ

99.85%

形状

foil

メーカー/製品名

Goodfellow 318-334-13

抵抗性

9.71 μΩ-cm

直径×厚み

6 mm × 0.008 mm

bp

2750 °C (lit.)

mp

1535 °C (lit.)

密度

7.86 g/mL at 25 °C (lit.)

SMILES記法

[Fe]

InChI

1S/Fe

InChI Key

XEEYBQQBJWHFJM-UHFFFAOYSA-N

詳細

For updated SDS information please visit www.goodfellow.com.

法的情報

Goodfellow製品

試験成績書(COA)

製品のロット番号・バッチ番号を入力して、試験成績書(COA) を検索できます。ロット番号・バッチ番号は、製品ラベルに「Lot」または「Batch」に続いて記載されています。

以前この製品を購入いただいたことがある場合

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文書ライブラリにアクセスする

Qiu et al.
Physical review letters, 85(7), 1492-1495 (2000-09-06)
FeF (2) films are grown by the reaction of XeF (2) and SeF (6) with iron foil. The growth initially follows the Mott-Cabrera parabolic rate law, indicating that the process is diffusion limited. At a certain film thickness, however, the
Arnold L Demain et al.
Applied microbiology and biotechnology, 73(1), 55-59 (2006-04-20)
When tetanus toxin is made by fermentation with Clostridium tetani, the traditional source of iron is an insoluble preparation called reduced iron powder. This material removes oxygen from the system by forming FeO(2) (rust). When inoculated in a newly developed
Jun-Won Jang et al.
Water science and technology : a journal of the International Association on Water Pollution Research, 59(12), 2503-2507 (2009-06-23)
Zero valent iron has been successfully used for the degradation of a wide range of contaminants. However, this reaction of using ZVI particle produces a large quantity of iron sludge. To solve the problem, we report the synthesis of self-organized
50 years ago in the Journal of Pediatrics: Iron metabolism in premature infants: II. Prevention of iron deficiency.
Pamela J Kling
The Journal of pediatrics, 164(4), 768-768 (2014-03-22)
Thomas A Russo et al.
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our

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