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Safety Information

N7132

Sigma-Aldrich

β-Nicotinamide adenine dinucleotide lithium salt from Saccharomyces cerevisiae

≥95%

Synonym(s):

β-DPN, β-NAD, Coenzyme 1, Cozymase, DPN, Diphosphopyridine nucleotide, NAD, Nadide

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500 MG
¥27,000
1 G
¥46,100

¥27,000

List Price¥33,800Save 20%

Estimated to ship onMarch 28, 2025

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500 MG
¥27,000
1 G
¥46,100

About This Item

Linear Formula:
C21H27N7O14P2Li
CAS Number:
64417-72-7
Molecular Weight:
670.37
EC Number:
264-884-1
MDL number:
MFCD00036974
UNSPSC Code:
41106305
PubChem Substance ID:
24897811
NACRES:
NA.51

¥27,000

List Price¥33,800Save 20%

Estimated to ship onMarch 28, 2025

Request a Bulk Order

Quality Level

200

Assay

≥95%

form

powder

storage temp.

−20°C

SMILES string

[Li].NC(=O)C1=CC=C[N](=C1)C2OC(COP(O)(=O)OP(O)(=O)OCC3OC(C(O)C3O)n4cnc5c(N)ncnc45)C(O)C2O

InChI

1S/C21H28N7O14P2.Li.H/c22-17-12-19(25-7-24-17)28(8-26-12)21-16(32)14(30)11(41-21)6-39-44(36,37)42-43(34,35)38-5-10-13(29)15(31)20(40-10)27-3-1-2-9(4-27)18(23)33;;/h1-4,7-8,10-11,13-16,20-21,29-32H,5-6H2,(H2,23,33)(H,34,35)(H,36,37)(H2,22,24,25);;

InChI key

CUAVZMNXWOTNRC-UHFFFAOYSA-N

Gene Information

human ... ESR1(2099) , ESR2(2100)

Application

β-Nicotinamide adenine dinucleotide (NAD+) and β-Nicotinamide adenine dinucleotide, reduced (NADH) comprise a coenzyme redox pair (NAD+:NADH) involved in a wide range of enzyme catalyzed oxidation reduction reactions. In addition to its redox function, NAD+/NADH is a donor of ADP-ribose units in ADP-ribosylaton (ADP-ribosyltransferases; poly(ADP-ribose) polymerases ) reactions and a precursor of cyclic ADP-ribose (ADP-ribosyl cyclases).

Biochem/physiol Actions

Electron acceptor

Other Notes

This is the common form of NAD.

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Regulatory Listings

Regulatory Listings are mainly provided for chemical products. Only limited information can be provided here for non-chemical products. No entry means none of the components are listed. It is the user’s obligation to ensure the safe and legal use of the product.

JAN Code

N7132-5G:
N7132-500MG:
N7132-VAR:
N7132-1G:
N7132-BULK:

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Certificates of Analysis (COA)

Lot/Batch Number
0000393198
0000393199
0000360237
0000280264
0000344541

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Michael O Hottiger
FEBS letters, 585(11), 1595-1599 (2011-03-23)
ADP-ribosylation is a covalent post-translational protein modification catalyzed by ADP-ribosyltransferases and is involved in important processes such as cell cycle regulation, DNA damage response, replication or transcription. Histones are ADP-ribosylated by ADP-ribosyltransferase diphtheria toxin-like 1 at specific amino acid residues
Richard P Ebstein et al.
FEBS letters, 585(11), 1529-1536 (2011-05-12)
Increasing evidence suggests that the nonapeptide, oxytocin (OT), helps shape social and affiliative behaviors not only in lower mammals but also in humans. Recently, an essential mediator of brain OT release has been discovered, ADP-ribosyl cyclase and/or CD38. We have
Lei Wang et al.
Veterinary microbiology, 177(1-2), 175-183 (2015-03-31)
Actinobacillus pleuropneumoniae is a causative agent of porcine pleuropneumonia, which is a highly contagious endemic disease of pigs. Adhesion is a critical first step in the infection process. Trimeric autotransporter adhesions (TAAs) have been identified as novel virulence factors; however
Katalin Völgyi et al.
Journal of proteomics, 120, 142-157 (2015-03-19)
The synapse is a particularly important compartment of neurons. To reveal its molecular characteristics we isolated whole brain synaptic (sMito) and non-synaptic mitochondria (nsMito) from the mouse brain with purity validated by electron microscopy and fluorescence activated cell analysis and
Junxiang Yin et al.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism, 35(11), 1783-1789 (2015-06-11)
Stroke is one of the leading causes of death. Growing evidence indicates that ketone bodies have beneficial effects in treating stroke, but their underlying mechanism remains unclear. Our previous study showed ketone bodies reduced reactive oxygen species by using NADH
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