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Amberlite IRN77 hydrogen form

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About This Item

UNSPSC Code:
23151817

form

beads

crosslinking

8 % cross-linked

packaging

bucket of 500 g

parameter

121 °C max. temp.

moisture

49-55%

technique(s)

LPLC: suitable

matrix

styrene-divinylbenzene (gel)

matrix active group

SCX

particle size

600-700 μm

operating pH

0-14

capacity

1.9 meq/mL by wetted bed volume

separation technique

cation exchange

General description

Amberlite IRN77 is a strong acid cation exchange resin.
As Nuclear Grade, a minimum of 99% of the available exchange sites are in the hydrogen form.
Strongly acidic cation exchange resin for water treatment, RAD waste treatment, decontamination.

Application

Amberlite IRN77 was used as cation exchanger to determine the sorption activity towards Cs(I) and Sr(II) using isothermal titration calorimetry and solution-depletion methods.

Legal Information

Amberlite is a trademark of DuPont de Nemours, Inc.

Pictograms

Exclamation mark

Signal Word

Warning

Hazard Statements

Precautionary Statements

Hazard Classifications

Eye Irrit. 2

Storage Class Code

10 - Combustible liquids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Benedicte Prelot et al.
Environmental science and pollution research international, 21(15), 9334-9343 (2014-04-15)
Sorption performance of cation-exchange resins Amberlite® IRN77 and Amberlite™ IRN9652 toward Cs(I) and Sr(II) has been tested in single-component aqueous solutions and simulated waste effluents containing other monovalent (Effluent 1) or divalent (Effluent 2) metal cations, as well as nitrate
Kyeong-Ho Yeon et al.
Water research, 38(7), 1911-1921 (2004-03-18)
This study investigated the production of high-purity water in the primary coolant of a nuclear power plant via the continuous electrodeionization (CEDI) process, using ion exchange resins as ion-conducting media between ion exchange membranes. The effectiveness of this method was
Cristina Morales Torres et al.
Nature communications, 11(1), 1792-1792 (2020-04-15)
Continuous cancer growth is driven by subsets of self-renewing malignant cells. Targeting of uncontrolled self-renewal through inhibition of stem cell-related signaling pathways has proven challenging. Here, we show that cancer cells can be selectively deprived of self-renewal ability by interfering

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