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799416

Sigma-Aldrich

Sodium nitrite

anhydrous, free-flowing, Redi-Dri, ACS reagent, ≥97%

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About This Item

Linear Formula:
NaNO2
CAS Number:
Molecular Weight:
69.00
EC Number:
MDL number:
UNSPSC Code:
12352302
PubChem Substance ID:
NACRES:
NA.21

grade

ACS reagent
anhydrous

Quality Level

vapor pressure

<0.0001 hPa ( 25 °C)

product line

Redi-Dri

Assay

≥97%

form

powder or crystals

autoignition temp.

914 °F

quality

free-flowing

impurities

≤0.01% insolubles

pH

9

mp

271 °C (lit.)

anion traces

chloride (Cl-): ≤0.005%
sulfate (SO42-): ≤0.01%

cation traces

Ca: ≤0.01%
Fe: ≤0.001%
K: ≤0.005%
heavy metals (as Pb): ≤0.001%

SMILES string

[Na+].[O-]N=O

InChI

1S/HNO2.Na/c2-1-3;/h(H,2,3);/q;+1/p-1

InChI key

LPXPTNMVRIOKMN-UHFFFAOYSA-M

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Application

Legal Information

Redi-Dri is a trademark of Sigma-Aldrich Co. LLC

Signal Word

Danger

Hazard Statements

Hazard Classifications

Acute Tox. 3 Oral - Aquatic Acute 1 - Eye Irrit. 2 - Ox. Sol. 3

Storage Class Code

5.1B - Oxidizing hazardous materials

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


Certificates of Analysis (COA)

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P J McClure et al.
International journal of food microbiology, 14(1), 77-91 (1991-10-01)
An automated turbidimetric system using multiwelled plates was used to examine the effects of different combinations of NaCl (0.5-8.0% w/v), NaNO2 (0-400 micrograms/ml) pH (4.6-7.4) and temperature (5-30 degrees C) on the growth of Listeria monocytogenes in tryptone soya broth.
R Kokkola et al.
Scandinavian journal of immunology, 61(1), 1-9 (2005-01-13)
Abstract High-mobility group box chromosomal protein 1 (HMGB1) is a protein with both intranuclear functions and extracellular cytokine-like effects. In this report, we study possible candidate receptors for HMGB1 on macrophages (Mphi) and define pathways activated by HMGB1 binding. Bone
Eagleson M.
Concise Encyclopedia Chemistry, 999-999 (1994)
Eagleson M.
Concise Encyclopedia Chemistry, 556-556 (1994)
Rustem F Dautov et al.
Nitric oxide : biology and chemistry, 40, 36-44 (2014-05-27)
Previous studies in non-human blood vessels and in platelets have demonstrated that under hypoxic conditions release of NO from nitrite (NO2(-)) is potentiated by deoxyhaemoglobin. In the current study, we characterized hypoxic potentiation of NO2(-) effects in human vasculature and

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