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690228

Sigma-Aldrich

N,N-Bis[3-(methylamino)propyl]methylamine

98%

Synonym(s):

N-Methyl-3,3′-bis(methylamino)dipropylamine, N-Methyl-N,N-bis[3-(methylamino)propyl]amine, Methylbis(3-methylaminopropyl)amine

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About This Item

Linear Formula:
CH3N(CH2CH2CH2NHCH3)2
CAS Number:
123-70-6
Molecular Weight:
173.30
Beilstein:
6711228
EC Number:
204-645-0
MDL number:
MFCD00014857
UNSPSC Code:
12352100
PubChem Substance ID:
329761321
NACRES:
NA.22

Assay

≥97.5% (GC)
97.5-102.5% (with HClO4, T)
98%

form

liquid

impurities

≤1% water

SMILES string

CNCCCN(C)CCCNC

InChI

1S/C9H23N3/c1-10-6-4-8-12(3)9-5-7-11-2/h10-11H,4-9H2,1-3H3

InChI key

PQYGBJHVVYREGU-UHFFFAOYSA-N

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Pictograms

Corrosion
GHS05

Signal Word

Danger

Hazard Statements

H314

Hazard Classifications

Skin Corr. 1B

Storage Class Code

8A - Combustible corrosive hazardous materials

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Certificates of Analysis (COA)

Lot/Batch Number
1414612V
1414612

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Gold thioglucose, mimpa and lesions of the ventromedial hypothalamus.
D F Brown
Journal of neuropathology and experimental neurology, 43(4), 455-457 (1984-07-01)
D F Brown et al.
Physiology & behavior, 49(1), 41-46 (1991-01-01)
It has been demonstrated that the ventromedial hypothalamus (VMH) of alloxan-induced diabetic mice is protected from subsequent gold thioglucose (GTG)-induced lesions. Another compound, 3,3'-methyliminobis-(N-methylpropylamine) (MIMPA), a triamine structurally unrelated to GTG, has been shown to cause similar VMH lesions in
J C Lyons et al.
Journal of neuropathology and experimental neurology, 41(1), 45-53 (1982-01-01)
The clinical and pathologic features of two brothers with the adult form of Gaucher's disease, both of whom developed cerebral glioblastoma multiforme, are presented. Neither patient had a long-standing neurologic disorder, and morphologic evidence of nervous system glucocerebroside storage was
S Levine et al.
Brain research, 242(2), 219-225 (1982-06-24)
An aliphatic triamine has been reported to cause lesions in rats in the vicinity of the area postrema and the median eminence of the hypothalamus, sites known to lack a blood-brain barrier. The present study revealed that some of the
S Levine et al.
Journal of immunological methods, 54(3), 355-360 (1982-11-12)
In the past, the lesions of experimental allergic encephalomyelitis (EAE) have been induced to localize around brain tissue damaged by anoxia or direct physical or chemical attack. The procedure for producing the requisite antecedent brain injury has been simplified by
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