Recognizes an epitope common to Glutamate Receptors 5, 6 and 7. No reactivity seen to GluR1, GluR2, GluR3 or GluR4.
Immunogen
Recombinant fusion protein TrpE-GluR5
Application
Anti-Glutamate Receptor 5, 6, 7 Antibody, clone 4F5 detects level of Glutamate Receptor 5 & has been published & validated for use in ELISA, IC, IH, RIA.
Immunohistochemistry on 4% paraformaldehyde fixed tissue:1:1,000-1:2,000 Immunocytochemistry
ELISA
RIA
Optimal working dilutions must be determined by end user.
Research Category Neuroscience
Research Sub Category Neurotransmitters & Receptors
Physical form
Ascites. Liquid. Contains no preservative.
Storage and Stability
Maintain at -20°C in undiluted aliquots for up 12 months after date of receipt. Avoid repeated freeze/thaw cycles.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Storage Class Code
10 - Combustible liquids
WGK
WGK 1
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
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Glutamate receptor (GluR) subunit composition of inferior salivatory nucleus (ISN) neurons was studied by immunohistochemical staining of retrogradely labeled neurons. Preganglionic ISN neurons innervating the von Ebner or parotid salivary glands were labeled by application of a fluorescent tracer to
Nesfatin-1, identified as an anorexigenic peptide, regulates the energy metabolism by suppressing food intake. The majority of nesfatin-1-synthesizing neurons are concentrated in various hypothalamic nuclei, especially in the supraoptic (SON), arcuate (ARC) and paraventricular nuclei (PVN). We tested the hypothesis
The dose-response relationship is a fundamental pharmacological parameter necessary to determine therapeutic thresholds. Epi-allelic hypomorphic analysis using RNA interference (RNAi) can similarly correlate target gene dosage with cellular phenotypes. This however requires a set of RNAi triggers empirically determined to
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