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Merck
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Key Documents

SAB4700676

Sigma-Aldrich

Monoclonal Anti-Blood Group B antibody produced in mouse

clone HEB-29, culture supernatant, buffered aqueous solution

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

biological source

mouse

Quality Level

100

conjugate

unconjugated

antibody form

culture supernatant

antibody product type

primary antibodies

clone

HEB-29, monoclonal

form

buffered aqueous solution

species reactivity

human

technique(s)

agglutination assay: suitable
immunohistochemistry: suitable

isotype

IgM

shipped in

wet ice

storage temp.

2-8°C

target post-translational modification

unmodified

General description

The antibody HEB-29 reacts with human blood group B. The specifity of the antibody HEB-29 was confirmed by comparison of specifity and reactivity to standard reagent using >5.000 samples of blood.

Immunogen

Mixture of erythrocytes of group B and glycoprotein fraction isolated from saliva of secretors with blood group B.

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Physical form

Solution in high glucose Dulbecco′s modified Eagle´s Medium (DMEM) with stable glutamine, with 15 mM sodium azide.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class

10 - Combustible liquids

wgk_germany

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificados de análisis (COA)

Busque Certificados de análisis (COA) introduciendo el número de lote del producto. Los números de lote se encuentran en la etiqueta del producto después de las palabras «Lot» o «Batch»

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Scientific reports, 10(1), 12871-12871 (2020-08-01)
Plasmodium falciparum causes the most severe form of malaria in humans. The adhesion of the infected erythrocytes (IEs) to endothelial receptors (sequestration) and to uninfected erythrocytes (rosetting) are considered major elements in the pathogenesis of the disease. Both sequestration and

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