Herbicide that prevents microtubule polymerization in plant cells, but not animal cells. Crop plants resistant to amiprofos-methyl and several other herbicides have been developed by insertion of a xenobiotic-detoxifying human gene CYP2B6.
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Arabinogalactan proteins (AGPs), a superfamily of plant hydroxyproline-rich glycoproteins, are present at cell surfaces. Although precise functions of AGPs remain elusive, they are widely implicated in plant growth and development. A well-characterized classical tomato (Lycopersicon esculentum) AGP containing a glycosylphosphatidylinositol
Herbicides and fungicides stimulate Ca2+ efflux from rat liver mitochondria.
Annals of the New York Academy of Sciences, 466, 767-784 (1986-01-01)
Mitosis is arrested in Haemanthus endosperm by amiprophos-methyl and oryzalin at a concentration of 100 nM, and anaphase chromosome movements are modified at 10 nM. Prolonged exposure to these drugs results in a classical c-mitosis. Anaphase chromosome movement is arrested
Three independent pleiotropic drug-resistance (pdr) mutants were isolated by selecting for resistance to the anti-microtubule herbicides amiprophos-methyl (APM) and oryzalin (ORY). These three mutants and a previously isolated mutant, ani1 (anisomycin resistance), were semi-dominant in heterozygous diploids, and they displayed
The Biochemical journal, 212(3), 567-572 (1983-06-15)
In maize mitochondria the effect of the herbicide amiprophosmethyl was studied on massive and limited Ca2+-loading and on mitochondrial energy transduction. Under massive Ca2+-loading conditions amiprophosmethyl inhibited the Ca2+ transport system directly without a significant effect on the respiratory chain
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