AB3230
Anti-Low Density Lipoprotein Antibody, copper oxidized
serum, Chemicon®
Synonyme(s) :
LDL
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About This Item
Code UNSPSC :
12352203
eCl@ss :
32160702
Nomenclature NACRES :
NA.41
Produits recommandés
Source biologique
rabbit
Niveau de qualité
Forme d'anticorps
serum
Type de produit anticorps
primary antibodies
Clone
polyclonal
Espèces réactives
human
Fabricant/nom de marque
Chemicon®
Technique(s)
ELISA: suitable
immunohistochemistry: suitable
western blot: suitable
Conditions d'expédition
wet ice
Modification post-traductionnelle de la cible
unmodified
Informations sur le gène
human ... LDLR(3949)
Spécificité
Reacts strongly to fully oxidatized modifications of LDL including Cu2+-oxidized LDL, MDA-LDL, HOCL-LDL but not to other oxidized proteins like MDA-HSA, HOCL-HSA, MDA-HDL, HOCL-HDL (below detection limit). The reaction of native LDL was weak, but clearly detectable (approx. 20%). Minimally oxidized LDL gave a strong binding signal (>80%).
Immunogène
Epitope: copper oxidized
Fully Cu2+-oxidized LDL.
Application
Anti-Low Density Lipoprotein Antibody, copper oxidized is an antibody against Low Density Lipoprotein for use in ELISA, WB, IH.
Research Category
Metabolism
Metabolism
Research Sub Category
Lipid Metabolism & Weight Regulation
Lipid Metabolism & Weight Regulation
Western Blot
Immunohistochemistry: 1:50-1:500
ELISA: 1:500-1:5,000
Optimal working dilutions must be determined by the end user.
Immunohistochemistry: 1:50-1:500
ELISA: 1:500-1:5,000
Optimal working dilutions must be determined by the end user.
Forme physique
Rabbit serum. Lyophilized. Contains no preservative. Reconstitute with 100 μL of sterile distilled water.
Stockage et stabilité
Maintain lyophilized material at 2-8°C for up to 12 months. After reconstitution maintain at -20°C in undiluted aliquots for up to 6 months. Avoid repeated freeze/thaw cycles.
Informations légales
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Clause de non-responsabilité
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Code de la classe de stockage
11 - Combustible Solids
Classe de danger pour l'eau (WGK)
WGK 1
Point d'éclair (°F)
Not applicable
Point d'éclair (°C)
Not applicable
Certificats d'analyse (COA)
Recherchez un Certificats d'analyse (COA) en saisissant le numéro de lot du produit. Les numéros de lot figurent sur l'étiquette du produit après les mots "Lot" ou "Batch".
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Retrouvez la documentation relative aux produits que vous avez récemment achetés dans la Bibliothèque de documents.
Malcolm R Ogborn et al.
Lipids, 43(9), 783-791 (2008-07-17)
Conjugated linoleic acid (CLA) is anti-proliferative and anti-inflammatory in the Han:SPRD-cy rat model of kidney disease. We used different doses of CLA and examined effects on renal histological benefit, the renal PPARgamma system and hepatic and renal levels of CLA
Dietary soya protein during pregnancy and lactation in rats with hereditary kidney disease attenuates disease progression in offspring.
Cahill, LE; Peng, CY; Bankovic-Calic, N; Sankaran, D; Ogborn, MR; Aukema, HM
The British Journal of Nutrition null
Deepa Sankaran et al.
Nephron. Experimental nephrology, 106(4), e122-e128 (2007-07-12)
Dietary soy protein and flax oil retard kidney disease progression when initiated in the early stages of disease in several experimental models, including the Han:SPRD-cy rat. However, individuals with kidney disease often do not become aware of their condition until
Deficiency in the metabolite receptor SUCNR1 (GPR91) leads to outer retinal lesions.
Favret, S; Binet, F; Lapalme, E; Leboeuf, D; Carbadillo, J; Rubic, T; Picard, E; Mawambo et al.
Aging null
Tadashi Uramatsu et al.
Biological & pharmaceutical bulletin, 36(8), 1271-1277 (2013-08-02)
Stroke-prone spontaneously hypertensive (SHRsp) rats develop severe hypertension resulting in renal injury. We investigated apoptosis inhibitor of macrophages (AIM) expression in nephrosclerotic rats and the involvement of AIM in olmesartan (OLM)- and azelnidipine (AZN)-induced decreases in the number of macrophages
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