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S9384

Supelco

Suloctidil

analytical standard, for drug analysis

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About This Item

Empirical Formula (Hill Notation):
C20H35NOS
CAS Number:
54767-75-8
Molecular Weight:
337.56
EC Number:
259-332-1
MDL number:
MFCD00238751
UNSPSC Code:
41116107
PubChem Substance ID:
329824622
NACRES:
NA.24

technique(s)

HPLC: suitable
gas chromatography (GC): suitable

application(s)

forensics and toxicology
pharmaceutical (small molecule)
veterinary

format

neat

storage temp.

2-8°C

SMILES string

CCCCCCCCN[C@H](C)[C@@H](O)c1ccc(SC(C)C)cc1

InChI

1S/C20H35NOS/c1-5-6-7-8-9-10-15-21-17(4)20(22)18-11-13-19(14-12-18)23-16(2)3/h11-14,16-17,20-22H,5-10,15H2,1-4H3/t17-,20-/m1/s1

InChI key

BFCDFTHTSVTWOG-YLJYHZDGSA-N

Application

Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.

Pictograms

Exclamation mark
GHS07

Signal Word

Warning

Hazard Statements

H302

Precautionary Statements

P264 - P270 - P301 + P312 - P501

Hazard Classifications

Acute Tox. 4 Oral

Storage Class Code

13 - Non Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Certificates of Analysis (COA)

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Combination of suloctidil and anticoagulation in the prevention of reocclusion after femoro-popliteal PTA.
F Mahler et al.
VASA. Zeitschrift fur Gefasskrankheiten, 16(4), 381-385 (1987-01-01)
P Kontro et al.
Neuroscience, 19(3), 1007-1010 (1986-11-01)
The effects of taurine and its structural analogues and two new anticonvulsant derivatives, taltrimide and MY-103, on the function of brain dopaminergic systems were studied by assessing their interference with the binding of [3H]spiperone to synaptic membranes isolated from rat
J M Boeynaems et al.
Biochemical pharmacology, 36(10), 1629-1635 (1987-05-15)
Suloctidil is a calcium antagonist with vascular relaxing activity and an antithrombotic agent: its antiplatelet action has been demonstrated in vivo, but is difficult to reproduce in vitro and the mechanism of this effect remains unknown. We have observed that
C Mazière et al.
Journal of clinical chemistry and clinical biochemistry. Zeitschrift fur klinische Chemie und klinische Biochemie, 26(1), 3-6 (1988-01-01)
Human foetal lung fibroblasts were pretreated for 24 h with the antithrombotic drug, suloctidil (1 to 10 mumol/l), which induced a dose-dependent increase in LDL binding, uptake and degradation. At 10 mumol/l suloctidil, the respective increases in these parameters were
P Calderon et al.
Archives internationales de pharmacodynamie et de therapie, 284(1), 101-113 (1986-11-01)
The vasorelaxing effect of suloctidil was evaluated in isolated rat and rabbit aorta and in isolated rabbit mesenteric and saphenous artery. Suloctidil inhibited contractions induced by increasing extracellular calcium in depolarized arteries, mainly in a competitive way. In the rat
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