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609765

Sigma-Aldrich

Sodium hydroxide-16O solution

20 wt. % in H216O, 99.9 atom % 16O

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About This Item

Formule linéaire :
Na16OH
Poids moléculaire :
39.99
Code UNSPSC :
12352302
ID de substance PubChem :
329759329

Description

18O-depleted

Pureté isotopique

99.9 atom % 16O

Concentration

20 wt. % in H216O

Changement de masse

depleted

Chaîne SMILES 

[16OH-].[Na+]

InChI

1S/Na.H2O/h;1H2/q+1;/p-1/i;1+0

Clé InChI

HEMHJVSKTPXQMS-QMVMUTFZSA-M

Conditionnement

This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.

Pictogrammes

Corrosion
GHS05

Mention d'avertissement

Danger

Mentions de danger

H290,H314

Classification des risques

Eye Dam. 1 - Met. Corr. 1 - Skin Corr. 1A

Code de la classe de stockage

8B - Non-combustible corrosive hazardous materials

Classe de danger pour l'eau (WGK)

WGK 1

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable

Équipement de protection individuelle

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter

Certificats d'analyse (COA)

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Horacio Cardenas et al.
Epigenetics, 9(11), 1461-1472 (2014-12-04)
A key step in the process of metastasis is the epithelial-to-mesenchymal transition (EMT). We hypothesized that epigenetic mechanisms play a key role in EMT and to test this hypothesis we analyzed global and gene-specific changes in DNA methylation during TGF-β-induced
Joshua S McLane et al.
Biophysical journal, 109(2), 249-264 (2015-07-23)
Mechanical properties of the tumor microenvironment have emerged as key factors in tumor progression. It has been proposed that increased tissue stiffness can transform stromal fibroblasts into carcinoma-associated fibroblasts. However, it is unclear whether the three to five times increase
Johannes Egerer et al.
The Journal of investigative dermatology, 135(10), 2368-2376 (2015-05-23)
Gerodermia osteodysplastica is a hereditary segmental progeroid disorder affecting skin, connective tissues, and bone that is caused by loss-of-function mutations in GORAB. The golgin, RAB6-interacting (GORAB) protein localizes to the Golgi apparatus and interacts with the small GTPase RAB6. In
Hélène O B Gautier et al.
Nature communications, 6, 8518-8518 (2015-10-07)
Myelin regeneration can occur spontaneously in demyelinating diseases such as multiple sclerosis (MS). However, the underlying mechanisms and causes of its frequent failure remain incompletely understood. Here we show, using an in-vivo remyelination model, that demyelinated axons are electrically active
Miki Wada et al.
Nucleic acids research, 42(12), 7851-7866 (2014-06-11)
During termination of translation in eukaryotes, a GTP-binding protein, eRF3, functions within a complex with the tRNA-mimicking protein, eRF1, to decode stop codons. It remains unclear how the tRNA-mimicking protein co-operates with the GTPase and with the functional sites on
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