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T3392

Sigma-Aldrich

Testosterone 3-(O-carboxymethyl)oxime: BSA

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About This Item

CAS Number:
10190-93-9
MDL number:
MFCD00132414
UNSPSC Code:
51111800
NACRES:
NA.77

Assay

≥98% (TLC)

Quality Level

200

form

lyophilized powder

drug control

regulated under CDSA - not available from Sigma-Aldrich Canada

extent of labeling

20-30 mol steroid per mol BSA

solubility

phosphate buffer: 1.90-2.10 mg/mL, clear to slightly hazy, colorless (pH 7.50)

shipped in

ambient

storage temp.

2-8°C

InChI

1S/C21H31NO4/c1-20-9-7-14(22-26-12-19(24)25)11-13(20)3-4-15-16-5-6-18(23)21(16,2)10-8-17(15)20/h11,15-18,23H,3-10,12H2,1-2H3,(H,24,25)/t15-,16-,17-,18-,20-,21-/m0/s1

InChI key

VDYLVWGBLQNNAW-ZKHIMWLXSA-N

Related Categories

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Guoxin Guo et al.
Molecular and cellular endocrinology, 503, 110671-110671 (2019-12-06)
Although hippocampus-derived androgens play an important role in hippocampal synaptic plasticity, studies at the cellular level have received relatively less attention. Furthermore, the underlying signalling pathways associated with synaptic plasticity remain unclear. Results of the present study demonstrated that testosterone
Yizhou Zhang et al.
Aging, 11(8), 2281-2294 (2019-04-22)
The non-genomic actions of androgen-induced synaptic plasticity have been extensively studied. However, the underlying mechanisms remain controversial. We recently found that testosterone-fetal bovine serum albumin (T-BSA), a cell membrane-impermeable complex, led to a rapid increase in the postsynaptic density 95
Sha Li et al.
Molecular and cellular endocrinology, 414, 82-90 (2015-07-15)
Testosterone (T), the principal androgen, and its metabolite, dihydrotestosterone (DHT), are known to mediate their effects through binding to intracellular androgen receptors (iARs). In addition to their well-known genomic effects, androgens rapidly alter neuronal excitability through a non-genomic pathway mediated

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