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F0127

Sigma-Aldrich

D-(−)-Fructose

≥99% (HPLC)

Synonym(s):

D-Levulose, Fruit sugar

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About This Item

Empirical Formula (Hill Notation):
C6H12O6
CAS Number:
57-48-7
Molecular Weight:
180.16
Beilstein:
1239004
EC Number:
200-333-3
MDL number:
MFCD00148910
UNSPSC Code:
12352201
PubChem Substance ID:
24894716
NACRES:
NA.21

biological source

corn

Quality Level

300

Assay

≥99% (HPLC)

form

powder

technique(s)

HPLC: suitable

impurities

≤0.05% Glucose (enzymatic)

color

colorless

useful pH range

5.0-7 (25 °C, 18 g/L)

mp

119-122 °C (dec.) (lit.)

solubility

water: 790 g/L at 20 °C

storage temp.

room temp

SMILES string

OC[C@@H](O)[C@@H](O)[C@H](O)C(=O)CO

InChI

1S/C6H12O6/c7-1-3(9)5(11)6(12)4(10)2-8/h3,5-9,11-12H,1-2H2/t3-,5-,6-/m1/s1

InChI key

BJHIKXHVCXFQLS-UYFOZJQFSA-N

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General description

D-fructose is a crucial ketohexose, which is commonly present in free form in fruit juices, musk, and honey accompanied by glucose. It is a constituent of raffinose, sucrose, and inulin. Fructose is formed by the hydrolysis of inulin.

Application

D-(-)-Fructose has been used:
  • as a supplement in modified Sabouraud (mSAB)/Sabouraud broth to culture Zygosaccharomyces bailii and to prepare ketchup to avoid the presence of any chemical preservatives
  • to prepare hypoosmotic fructose solution to assess the functional integrity of sperm membranes
  • to supplement drinking water to mimic high-fructose corn syrup containing soft drinks

Other Notes

To gain a comprehensive understanding of our extensive range of Monosaccharides for your research, we encourage you to visit our Carbohydrates Category page.

Storage Class Code

11 - Combustible Solids

WGK

WGK 1

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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The regulation of hepatic gene expression has been extensively studied at the transcriptional level; however, the control of metabolism through posttranscriptional gene regulation by RNA-binding proteins in physiological and disease states is less understood. Here, we report a major role
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