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Key Documents

MAB329

Sigma-Aldrich

Anti-Synaptophysin Antibody, clone SP15

ascites fluid, clone SP15, Chemicon®

Synonyme(s) :

Major Synaptic Vesicle Protein p38

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About This Item

Code UNSPSC :
12352203
eCl@ss :
32160702
Nomenclature NACRES :
NA.41

Source biologique

mouse

Niveau de qualité

Forme d'anticorps

ascites fluid

Type de produit anticorps

primary antibodies

Clone

SP15, monoclonal

Espèces réactives

pig, rat, hamster, human

Fabricant/nom de marque

Chemicon®

Technique(s)

ELISA: suitable
immunohistochemistry: suitable (paraffin)
western blot: suitable

Isotype

IgM

Numéro d'accès NCBI

Numéro d'accès UniProt

Conditions d'expédition

dry ice

Modification post-traductionnelle de la cible

unmodified

Informations sur le gène

human ... SYP(6855)

Spécificité

Synaptophysin. Reacts with synaptophysin expressed in CHO cells.

Immunogène

Crude synaptic immunoprecipitate (Human)

Application

Immunohistochemistry at 1:200-1:5,000.

Formalin fixed (fixed <4 weeks), paraffin embedded

Frozen

4% Paraformaldehyde

Western blotting at 1:2,500-1:10,000.

ELISA at 1:500

Optimal working dilutions must be determined by the end user.
Research Category
Neuroscience
Research Sub Category
Synapse & Synaptic Biology
This Anti-Synaptophysin Antibody, clone SP15 is validated for use in ELISA, WB, IH(P) for the detection of Synaptophysin.

Liaison

Replaces: 04-1019

Forme physique

Liquid

Stockage et stabilité

Maintain frozen at -20°C in undiluted aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.

Informations légales

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Clause de non-responsabilité

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Code de la classe de stockage

10 - Combustible liquids

Classe de danger pour l'eau (WGK)

WGK 1

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable


Certificats d'analyse (COA)

Recherchez un Certificats d'analyse (COA) en saisissant le numéro de lot du produit. Les numéros de lot figurent sur l'étiquette du produit après les mots "Lot" ou "Batch".

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Retrouvez la documentation relative aux produits que vous avez récemment achetés dans la Bibliothèque de documents.

Consulter la Bibliothèque de documents

Gerald D Griffin et al.
The Journal of comparative neurology, 518(22), 4531-4545 (2010-10-05)
Central oxytocin (OT) modulates many social behaviors, including female rat sexual receptivity, quantified as the copulatory stance known as lordosis. The expression of the lordosis response is modulated by OT action in the ventromedial nucleus of the hypothalamus (VMH), as
Michelle A Land et al.
Scientific reports, 9(1), 16737-16737 (2019-11-15)
A non-synonymous single nucleotide polymorphism of the human serotonin 5-HT2C receptor (5-HT2CR) gene that converts a cysteine to a serine at amino acid codon 23 (Cys23Ser) appears to impact 5-HT2CR pharmacology at a cellular and systems level. We hypothesized that
Raymond S Roginski et al.
Gene, 648, 42-53 (2018-01-18)
The known functions of the human GCOM1 complex hub gene include transcription elongation and the intercalated disk of cardiac myocytes. However, in all likelihood, the gene's most interesting, and thus far least understood, roles will be found in the central
A Odawara et al.
Scientific reports, 6, 26181-26181 (2016-05-18)
The functional network of human induced pluripotent stem cell (hiPSC)-derived neurons is a potentially powerful in vitro model for evaluating disease mechanisms and drug responses. However, the culture time required for the full functional maturation of individual neurons and networks
Luca Bragina et al.
Journal of neurochemistry, 99(1), 134-141 (2006-09-22)
In rat frontal cortex, extracellular levels of glutamate are raised by the anti-psychotic drug clozapine. We have recently shown that a significant reduction in the levels of the glutamate transporter GLT-1 may be one of the mechanisms responsible for this

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