GF15991140
Iron
foil, 50x50mm, thickness 0.1mm, hard, 99.5%
Synonyme(s) :
Iron, FE000320
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About This Item
Produits recommandés
Pureté
99.50%
Forme
foil
Fabricant/nom de marque
Goodfellow 159-911-40
Résistivité
9.71 μΩ-cm
Taille × épaisseur
50 x 50 mm × 0.1 mm
Point d'ébullition
2750 °C (lit.)
Pf
1535 °C (lit.)
Densité
7.86 g/mL at 25 °C (lit.)
Chaîne SMILES
[Fe]
InChI
1S/Fe
Clé InChI
XEEYBQQBJWHFJM-UHFFFAOYSA-N
Description générale
For updated SDS information please visit www.goodfellow.com.
Informations légales
Product of Goodfellow
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Physical review letters, 85(7), 1492-1495 (2000-09-06)
FeF (2) films are grown by the reaction of XeF (2) and SeF (6) with iron foil. The growth initially follows the Mott-Cabrera parabolic rate law, indicating that the process is diffusion limited. At a certain film thickness, however, the
Applied microbiology and biotechnology, 73(1), 55-59 (2006-04-20)
When tetanus toxin is made by fermentation with Clostridium tetani, the traditional source of iron is an insoluble preparation called reduced iron powder. This material removes oxygen from the system by forming FeO(2) (rust). When inoculated in a newly developed
Infection and immunity, 82(6), 2203-2210 (2014-03-20)
In order to survive inside macrophages, Salmonella produces a series of proteins encoded by genes within Salmonella pathogenicity island 2 (SPI-2). In the present study, we report that Fur, a central regulator of iron utilization, negatively controls the expression of
Proceedings of the National Academy of Sciences of the United States of America, 111(14), 5171-5176 (2014-04-08)
The enzymes PhnY and PhnZ comprise an oxidative catabolic pathway that enables marine bacteria to use 2-aminoethylphosphonic acid as a source of inorganic phosphate. PhnZ is notable for catalyzing the oxidative cleavage of a carbon-phosphorus bond using Fe(II) and dioxygen
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our
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