Benzoyl peroxide (B5907) is specifically formulated as an initiator for these resins.
LR White Resin is a very low viscosity resin, non-toxic and suitable for various applications in both light and Electron Microscopy. Major advantage of LR White sections is that they show minimal non-specific staining. Sections of polymerized resin are hydrophilic, thus immunocytochemistry reagents easily penetrate into the section. Its low viscosity makes LR White an ideal tool for infiltrating plant tissues as well as decalcified bone and teeth.
Understanding which hydraulic traits are under genetic control and/or are phenotypically plastic is essential in understanding how tree species will respond to rapid shifts in climate. We quantified hydraulic traits in Eucalyptus obliqua L'Her. across a precipitation gradient in the
Development (Cambridge, England), 147(1) (2019-12-13)
Cilia rotation-driven nodal flow is crucial for the left-right (L-R) break in symmetry in most vertebrates. However, the mechanism by which the flow signal is translated to asymmetric gene expression has been insufficiently addressed. Here, we show that Hedgehog (Hh)
The Histochemical journal, 30(2), 105-109 (1999-04-07)
An improved new method for the simultaneous visualization of mRNA and encoded protein in LR White resin-embedded specimens is described. This pre-embedding electron microscopical in situ hybridization (procedure) localized rat growth hormone mRNA specifically as high electron-density products on the
Journal of electron microscopy, 58(4), 267-279 (2009-04-01)
We have developed a new standardized method for the post-embedding immunoelectron microscopy using the same fixation, antigen retrieval and image contrasting procedures. Tissues were fixed with 4% formaldehyde containing 2.5 mM CaCl(2), 1.25 mM MgCl(2) in a 0.1 M 4-(2-hydroxyethyl)-piperazineethanesulfonic
Neurobiology of disease, 114, 140-152 (2018-03-06)
Intracellular deposition of pathologically altered α-synuclein mostly in neurons characterises Parkinson's disease (PD), while its accumulation predominantly in oligodendrocytes is a feature of multiple system atrophy (MSA). Recently a prion-like spreading of pathologic α-synuclein has been suggested to play a
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