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SAB4100221

Sigma-Aldrich

Monoclonal Anti-PECR antibody produced in mouse

clone PECR-B172, culture supernatant

Synonym(s):

AntiDCRRP, AntiHPDHASE, AntiHSA250303, AntiPVIARL, AntiPeroxisomal trans-2-enoyl-CoA reductase, AntiSDR29C1, AntiTERP

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

biological source

mouse

conjugate

unconjugated

antibody form

culture supernatant

antibody product type

primary antibodies

clone

PECR-B172, monoclonal

form

aqueous solution

mol wt

antigen predicted mol wt 32.4 kDa

species reactivity

human

technique(s)

western blot: suitable

isotype

IgG1

UniProt accession no.

Q9BY49

shipped in

dry ice

storage temp.

−20°C

Gene Information

human ... PECR(55825)

Immunogen

PECR purified recombinant protein

Physical form

Solution in Dulbecco′s Modified Eagle′s Medium with 10% fetal calf serum and 15 mM sodium azide.

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Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Genetics of alcohol dependence
KImura M and Higuchi S
Psychiatry and Clinical Neurosciences, 65(3), 213-225 (2011)
J Gloerich et al.
FEBS letters, 580(8), 2092-2096 (2006-03-21)
Phytol is a naturally occurring precursor of phytanic acid. The last step in the conversion of phytol to phytanoyl-CoA is the reduction of phytenoyl-CoA mediated by an, as yet, unidentified enzyme. A candidate for this reaction is a previously described
A K Das et al.
The Journal of biological chemistry, 275(32), 24333-24340 (2000-05-16)
Chain elongation of fatty acids is an important cellular process and is believed to occur in the endoplasmic reticulum of all eukaroytic cells. Herein we describe the cloning and characterization of a peroxisomal NADPH-specific trans-2-enoyl-CoA reductase, the key enzyme for
Rajani Dinavahi et al.
Journal of the American Society of Nephrology : JASN, 22(6), 1168-1178 (2011-05-14)
Although T and B cell alloimmunity contribute to transplant injury, autoimmunity directed at kidney-expressed, non-HLA antigens may also participate. Because the specificity, prevalence, and importance of antibodies to non-HLA antigens in late allograft injury are poorly characterized, we used a

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