208880
Anti-Calnexin, C-Terminal (575-593) Rabbit pAb
liquid, Calbiochem®
Synonym(s):
Anti-IP90, Anti-p88
Sign Into View Organizational & Contract Pricing
All Photos(1)
About This Item
UNSPSC Code:
12352203
NACRES:
NA.41
Recommended Products
biological source
rabbit
Quality Level
antibody form
purified antibody
antibody product type
primary antibodies
clone
polyclonal
form
liquid
contains
≤0.1% sodium azide as preservative
species reactivity
human, rabbit, mouse, rat, canine, guinea pig, bovine, hamster, monkey, sheep
manufacturer/tradename
Calbiochem®
storage condition
OK to freeze
avoid repeated freeze/thaw cycles
isotype
IgG
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
unmodified
General description
Anti-Calnexin, C-Terminal (575-593), rabbit polyclonal, recognizes the ~90 kDa calnexin protein. It is validated for Western blotting.
Protein A purified rabbit polyclonal antibody. Recognizes the ~90 kDa calnexin protein.
Recognizes the ~90 kDa calnexin protein.
Immunogen
Canine
a synthetic peptide (AEEDEILNRSPRNRKPRRE) corresponding to amino acids 575-593 of canine calnexin, conjugated to KLH
Application
Immunoblotting (1 µg/ml)
Warning
Toxicity: Standard Handling (A)
Physical form
In PBS, 50% glycerol, pH 7.2.
Reconstitution
Following initial thaw, aliquot and freeze (-20°C).
Analysis Note
Positive Control
Heat-shocked HeLa cells
Heat-shocked HeLa cells
Other Notes
Hammond, C., et al. 1994. J. Cell Biol. 126, 41.
Ou, W.-J., et al. 1993. Nature 364, 771.
David, V., et al. 1993. J. Biol. Chem.268, 9585.
Ahluwalia, N., et al. 1992. J. Biol. Chem.267, 10914.
Galvin, K., et al. 1992. Proc. Natl. Acad. Sci. USA89, 8452.
Hochstenbach, F., et al. 1992. Proc. Natl. Acad. Sci. USA89, 4734.
Wada, I., et al. 1991. J. Biol. Chem. 266, 19599.
Ou, W.-J., et al. 1993. Nature 364, 771.
David, V., et al. 1993. J. Biol. Chem.268, 9585.
Ahluwalia, N., et al. 1992. J. Biol. Chem.267, 10914.
Galvin, K., et al. 1992. Proc. Natl. Acad. Sci. USA89, 8452.
Hochstenbach, F., et al. 1992. Proc. Natl. Acad. Sci. USA89, 4734.
Wada, I., et al. 1991. J. Biol. Chem. 266, 19599.
Variables associated with assay conditions will dictate the optimal working dilution.
Legal Information
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
Not finding the right product?
Try our Product Selector Tool.
Storage Class Code
10 - Combustible liquids
WGK
WGK 1
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
Already Own This Product?
Find documentation for the products that you have recently purchased in the Document Library.
Claudia M Wunderlich et al.
Journal of immunology (Baltimore, Md. : 1950), 188(9), 4141-4144 (2012-04-03)
Circulating IL-6 levels correlate with the severity of blood-stage malaria in humans and mouse models, but the impact of IL-6 classic signaling through membrane IL-6Rα, as well as IL-6 trans-signaling through soluble IL-6Rα, on the outcome of malaria has remained
Jin Zhang et al.
Virology journal, 9, 303-303 (2012-12-12)
Although the aggregation of PrPSc is thought to be crucial for the neuropathology of prion diseases, there is evidence in cultured cells and transgenic mice that neuronal death can be triggered by the accumulation of cytosolic PrPs, leading to the
Martin Whitham et al.
The Journal of biological chemistry, 287(14), 10771-10779 (2012-02-22)
Exercise increases the expression of the prototypical myokine IL-6, but the precise mechanism by which this occurs has yet to be identified. To mimic exercise conditions, C2C12 myotubes were mechanically stimulated via electrical pulse stimulation (EPS). We compared the responses
Shami Kanekar et al.
Journal of neuroscience research, 87(6), 1424-1434 (2008-12-31)
One premise regarding the mechanism of injury-evoked neuroregeneration is that injured cells induce the release of neurotrophic factors to trigger neurogenesis. Extracellular purine nucleotides exert multiple neurotrophic actions in the central nervous system mediated via activation of purinergic receptors. However
Carol Ann Remme et al.
Circulation, 114(24), 2584-2594 (2006-12-06)
Patients carrying the cardiac sodium channel (SCN5A) mutation 1795insD show sudden nocturnal death and signs of multiple arrhythmia syndromes including bradycardia, conduction delay, QT prolongation, and right precordial ST-elevation. We investigated the electrophysiological characteristics of a transgenic model of the
Our team of scientists has experience in all areas of research including Life Science, Material Science, Chemical Synthesis, Chromatography, Analytical and many others.
Contact Technical Service