735949
Gadolinium(III) chloride
anhydrous, beads, −10 mesh, 99.99% trace metals basis
Synonym(s):
Gadolinium trichloride
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About This Item
Linear Formula:
GdCl3
CAS Number:
Molecular Weight:
263.61
EC Number:
MDL number:
UNSPSC Code:
12352302
PubChem Substance ID:
NACRES:
NA.23
Assay:
99.99% trace metals basis
grade:
anhydrous
form:
beads
Recommended Products
grade
anhydrous
Quality Level
Assay
99.99% trace metals basis
form
beads
particle size
−10 mesh
SMILES string
Cl[Gd](Cl)Cl
InChI
1S/3ClH.Gd/h3*1H;/q;;;+3/p-3
InChI key
MEANOSLIBWSCIT-UHFFFAOYSA-K
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Application
Gadolinium Chloride is a commonly used precursor for the formation of MRI contrast agent and can be used as a Diels-Alder catalyst.
Signal Word
Warning
Hazard Statements
Precautionary Statements
Hazard Classifications
Skin Irrit. 2
Storage Class Code
13 - Non Combustible Solids
WGK
WGK 3
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
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Xu, J.; et al.
Journal of the American Chemical Society, 7, 7245-7246 (1995)
Ma, Y.; et al.
The Journal of Organic Chemistry, 64, 6462-6467 (1999)
Puja Gulati et al.
Naunyn-Schmiedeberg's archives of pharmacology, 386(3), 255-264 (2012-12-12)
The present study was designed to investigate the potential of gadolinium, a stretch-activated calcium channel blocker in ischemic reperfusion (I/R)-induced brain injury in mice. Bilateral carotid artery occlusion of 12 min followed by reperfusion for 24 h was given to induce cerebral
Lillian Zhang et al.
The Journal of toxicological sciences, 37(2), 447-453 (2012-04-03)
Liver toxicity is a side effect observed with some herbal treatments, including Piper methysticum. The possible mechanisms responsible include inflammation subsequent to activation of liver macrophages and oxidative damage. Hepatotoxicity of the pharmacologically active component of Piper methysticum (kavalactones) was
Chikara Abe et al.
Journal of applied physiology (Bethesda, Md. : 1985), 114(1), 28-36 (2012-11-10)
Water drinking is known to induce the pressor response. The efferent pathway in this response involves sympathoexcitation, because the pressor response was completely abolished by ganglionic blockade or an α(1)-adrenergic antagonist. However, the afferent pathway in this response has not
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