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MAB5628

Sigma-Aldrich

Anti-Glial Fibrillary Acidic Protein Antibody

Chemicon®, from mouse

Synonym(e):

GFAP

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About This Item

UNSPSC-Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

Biologische Quelle

mouse

Qualitätsniveau

100

Antikörperform

purified immunoglobulin

Antikörper-Produkttyp

primary antibodies

Klon

monoclonal

Speziesreaktivität

rat, human

Hersteller/Markenname

Chemicon®

Methode(n)

immunocytochemistry: suitable
western blot: suitable

Isotyp

IgG2a

NCBI-Hinterlegungsnummer

NM_002055.2

UniProt-Hinterlegungsnummer

P14136

Versandbedingung

wet ice

Posttranslationale Modifikation Target

unmodified

Angaben zum Gen

human ... GFAP(2670)

Verwandte Kategorien

Anwendung

Detect Glial Fibrillary Acidic Protein using this Anti-Glial Fibrillary Acidic Protein Antibody validated for use in WB & IC.
Western blot: 1:10,000 on rat brain

Optimal working dilutions must be determined by end user.

Verlinkung

Replaces: 04-1031; 04-1062

Physikalische Form

Format: Purified
Purified immunoglobulin. Liquid in 0.02M phosphate buffer, 0.25 M NaCl, pH 7.6 with 0.1% sodium azide.

Hinweis zur Analyse

Control
Positive: Neural tissue or astrocyte culture
Negative: Non-neural tissue or neuronal culture

Sonstige Hinweise

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

Rechtliche Hinweise

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Lagerklassenschlüssel

10 - Combustible liquids

WGK

WGK 2

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

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Die Dokumentenbibliothek aufrufen

Takashi Ishijima et al.
Science progress, 104(4), 368504211054985-368504211054985 (2021-11-26)
By using an animal model in which inflammatory cytokines are induced in lipopolysaccharide (LPS)-injected rat brain, we investigated the induction of tumor necrosis factor alpha (TNFα), interleukin-1beta (IL-1β), and IL-6. Immunoblotting and immunohistochemistry revealed that all three cytokines were transiently
Shun-Yun Cheng et al.
Molecular therapy. Nucleic acids, 35(1), 102088-102088 (2024-01-09)
Inherited retinal dystrophies caused by dominant mutations in photoreceptor (PR) cell expressed genes are a major cause of irreversible vision loss. Oligonucleotide therapy has been of interest in diseases that conventional medicine cannot target. In the early days, small interfering
Sitthisak Thongrong et al.
Biomedical reports, 18(3), 20-20 (2023-02-18)
Chronic stress has been recognized to induce the alterations of neuronal and glial cells in the hippocampus, and is thus implicated in cognitive dysfunction. There is increasing evidence to indicate that natural compounds capable of exerting neuroprotective and antioxidant activities
Heather R Minkel et al.
Glia, 63(12), 2285-2297 (2015-07-21)
Alexander Disease (AxD) is a "gliopathy" caused by toxic, dominant gain-of-function mutations in the glial fibrillary acidic protein (GFAP) gene. Two distinct types of AxD exist. Type I AxD affected individuals develop cerebral symptoms by 4 years of age and
Jian-Ying Tian et al.
Experimental and therapeutic medicine, 12(2), 683-689 (2016-07-23)
The aim of the present study was to observe the effects of a general extract of Lycium bararum polysaccharides (LBPs) on methylmercury (MeHg)-induced damage in hippocampus neural stem cells (hNSCs). The hippocampal tissues of embryonic day 16 Sprague-Dawley rats were

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