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576794

Sigma-Aldrich

Ammonium-14N chloride

99.99 atom % 14N, 15N-depleted, 99% (CP)

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About This Item

Linear Formula:
14NH4Cl
Molecular Weight:
53.49
EC Number:
UNSPSC Code:
12352101
PubChem Substance ID:

isotopic purity

99.99 atom % 14N

Assay

99% (CP)

form

solid

mp

340 °C (subl.) (lit.)

mass shift

depleted

SMILES string

[14NH4+].[Cl-]

InChI

1S/ClH.H3N/h1H;1H3/i;1+0

InChI key

NLXLAEXVIDQMFP-QMVMUTFZSA-N

Packaging

This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.

Pictograms

Exclamation mark

Signal Word

Warning

Hazard Statements

Hazard Classifications

Acute Tox. 4 Oral - Eye Irrit. 2

Storage Class Code

13 - Non Combustible Solids

WGK

WGK 1

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Jackie Perrin et al.
Journal of cell science, 128(13), 2269-2277 (2015-05-23)
TM9 family proteins (also named Phg1 proteins) have been previously shown to control cell adhesion by determining the cell surface localization of adhesion proteins such as the Dictyostelium SibA protein. Here, we show that the glycine-rich transmembrane domain (TMD) of
Allison L Boyd et al.
The Journal of experimental medicine, 211(10), 1925-1935 (2014-09-03)
Allogeneic hematopoietic stem cell (HSC) transplantation (HSCT) is currently the leading strategy to manage acute myeloid leukemia (AML). However, treatment-related morbidity limits the patient generalizability of HSCT use, and the survival of leukemic stem cells (LSCs) within protective areas of
Christine Burkard et al.
PloS one, 9(7), e101762-e101762 (2014-07-16)
Studies of viral entry into host cells often rely on the detection of post-entry parameters, such as viral replication or the expression of a reporter gene, rather than on measuring entry per se. The lack of assays to easily detect
Sewa Rijal et al.
Blood, 125(18), 2815-2824 (2015-03-05)
Phosphoinositide signaling regulates diverse cellular functions. Phosphoinositide-3 kinase (PI3K) generates PtdIns(3,4,5)P3 and PtdIns(3,4)P2, leading to the activation of proliferative and anti-apoptotic signaling pathways. Termination of phosphoinositide signaling requires hydrolysis of inositol ring phosphate groups through the actions of PtdIns(3,4,5)P3 3-phosphatase
Pontus Lundberg et al.
The Journal of experimental medicine, 211(11), 2213-2230 (2014-10-08)
The majority of patients with myeloproliferative neoplasms (MPNs) carry a somatic JAK2-V617F mutation. Because additional mutations can precede JAK2-V617F, it is questioned whether JAK2-V617F alone can initiate MPN. Several mouse models have demonstrated that JAK2-V617F can cause MPN; however, in

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