MAB336
Anti-Syntaxin Antibody, clone SP8
ascites fluid, clone SP8, Chemicon®
Synonym(s):
Syntaxin 1A, Neuron-specific Antigen HPC-1, STX1A
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About This Item
UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41
Recommended Products
biological source
mouse
Quality Level
antibody form
ascites fluid
antibody product type
primary antibodies
clone
SP8, monoclonal
species reactivity
hamster, human, pig, rat
manufacturer/tradename
Chemicon®
technique(s)
ELISA: suitable
immunohistochemistry: suitable
western blot: suitable
isotype
IgG1
NCBI accession no.
UniProt accession no.
shipped in
dry ice
target post-translational modification
unmodified
Gene Information
human ... STX1A(6804)
Specificity
Syntaxin (34-36 kDa). Reacts with syntaxin expressed in bacterial systems.
Immunogen
Crude synaptic immunoprecipitate (Human).
Application
Immunohistochemistry at >1:100-1:200.
Formalin fixed (fixed <4 weeks)
Western blotting at 1:100-1:10,000.
ELISA at 1:500
Optimal working dilutions must be determined by the end user.
Formalin fixed (fixed <4 weeks)
Western blotting at 1:100-1:10,000.
ELISA at 1:500
Optimal working dilutions must be determined by the end user.
Research Category
Neuroscience
Neuroscience
Research Sub Category
Synapse & Synaptic Biology
Synapse & Synaptic Biology
This Anti-Syntaxin Antibody, clone SP8 is validated for use in ELISA, WB, IH for the detection of Syntaxin.
Physical form
Liquid with 0.1% Sodium Azide.
Storage and Stability
Maintain frozen at -20°C in undiluted aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Storage Class Code
12 - Non Combustible Liquids
WGK
nwg
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
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The gene for soluble N-ethylmaleimide sensitive factor attachment protein alpha is mutated in hydrocephaly with hop gait (hyh) mice.
Hong, HK; Chakravarti, A; Takahashi, JS
Proceedings of the National Academy of Sciences of the USA null
Liza Barki-Harrington et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 29(29), 9219-9226 (2009-07-25)
We aimed to test whether tyrosine phosphorylation of the NMDA receptor (NMDAR) in the insular cortex is necessary for novel taste learning. We found that in rats, novel taste learning leads to elevated phosphorylation of tyrosine 1472 of the NR2B
S Liu et al.
Neuroscience, 146(4), 1677-1688 (2007-05-01)
Serotonin (5-HT) action via the 5-HT(2C) receptor (5-HT(2C)R) provides an important modulatory influence over neurons of the prefrontal cortex (PFC), which is critically involved in disorders of executive function including substance use disorders. In the present study, we investigated the
P Osten et al.
Neuron, 21(1), 99-110 (1998-08-11)
In this study, we demonstrate specific interaction of the GluR2 alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA) receptor subunit C-terminal peptide with an ATPase N-ethylmaleimide-sensitive fusion protein (NSF) and alpha- and beta-soluble NSF attachment proteins (SNAPs), as well as dendritic colocalization of these proteins. The
Elizabeth Scarr et al.
Clinical psychopharmacology and neuroscience : the official scientific journal of the Korean College of Neuropsychopharmacology, 10(1), 25-33 (2013-02-23)
Given the ability of mood stabilizers and antipsychotics to promote cell proliferation, we wanted to determine the effects of these drugs on neuronal markers previously reported to be altered in subjects with psychiatric disorders. Male Sprauge-Dawley rats were treated with
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