Saltar al contenido
Merck
Todas las fotos(1)

Documentos clave

208880

Sigma-Aldrich

Anti-Calnexin, C-Terminal (575-593) Rabbit pAb

liquid, Calbiochem®

Sinónimos:

Anti-IP90, Anti-p88

Iniciar sesiónpara Ver la Fijación de precios por contrato y de la organización


About This Item

Código UNSPSC:
12352203
NACRES:
NA.41

origen biológico

rabbit

Nivel de calidad

forma del anticuerpo

purified antibody

tipo de anticuerpo

primary antibodies

clon

polyclonal

Formulario

liquid

contiene

≤0.1% sodium azide as preservative

reactividad de especies

human, rabbit, mouse, rat, canine, guinea pig, bovine, hamster, monkey, sheep

fabricante / nombre comercial

Calbiochem®

condiciones de almacenamiento

OK to freeze
avoid repeated freeze/thaw cycles

isotipo

IgG

Condiciones de envío

wet ice

temp. de almacenamiento

−20°C

modificación del objetivo postraduccional

unmodified

Descripción general

Anti-Calnexin, C-Terminal (575-593), rabbit polyclonal, recognizes the ~90 kDa calnexin protein. It is validated for Western blotting.
Protein A purified rabbit polyclonal antibody. Recognizes the ~90 kDa calnexin protein.
Recognizes the ~90 kDa calnexin protein.

Inmunógeno

Canine
a synthetic peptide (AEEDEILNRSPRNRKPRRE) corresponding to amino acids 575-593 of canine calnexin, conjugated to KLH

Aplicación

Immunoblotting (1 µg/ml)

Advertencia

Toxicity: Standard Handling (A)

Forma física

In PBS, 50% glycerol, pH 7.2.

Reconstitución

Following initial thaw, aliquot and freeze (-20°C).

Nota de análisis

Positive Control
Heat-shocked HeLa cells

Otras notas

Hammond, C., et al. 1994. J. Cell Biol. 126, 41.
Ou, W.-J., et al. 1993. Nature 364, 771.
David, V., et al. 1993. J. Biol. Chem.268, 9585.
Ahluwalia, N., et al. 1992. J. Biol. Chem.267, 10914.
Galvin, K., et al. 1992. Proc. Natl. Acad. Sci. USA89, 8452.
Hochstenbach, F., et al. 1992. Proc. Natl. Acad. Sci. USA89, 4734.
Wada, I., et al. 1991. J. Biol. Chem. 266, 19599.
Variables associated with assay conditions will dictate the optimal working dilution.

Información legal

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

¿No encuentra el producto adecuado?  

Pruebe nuestro Herramienta de selección de productos.

Código de clase de almacenamiento

10 - Combustible liquids

Clase de riesgo para el agua (WGK)

WGK 1


Certificados de análisis (COA)

Busque Certificados de análisis (COA) introduciendo el número de lote del producto. Los números de lote se encuentran en la etiqueta del producto después de las palabras «Lot» o «Batch»

¿Ya tiene este producto?

Encuentre la documentación para los productos que ha comprado recientemente en la Biblioteca de documentos.

Visite la Librería de documentos

Anita L Pinner et al.
Schizophrenia research, 177(1-3), 78-87 (2016-02-16)
Recent reports suggest abnormalities of neurotransmitter receptor trafficking, targeting, dendritic localization, recycling, and degradation in the brain in schizophrenia. We hypothesized that a potential explanation for these findings may be abnormal posttranslational modifications that influence intracellular targeting and trafficking of
Jin Zhang et al.
Virology journal, 9, 303-303 (2012-12-12)
Although the aggregation of PrPSc is thought to be crucial for the neuropathology of prion diseases, there is evidence in cultured cells and transgenic mice that neuronal death can be triggered by the accumulation of cytosolic PrPs, leading to the
Claudia M Wunderlich et al.
Journal of immunology (Baltimore, Md. : 1950), 188(9), 4141-4144 (2012-04-03)
Circulating IL-6 levels correlate with the severity of blood-stage malaria in humans and mouse models, but the impact of IL-6 classic signaling through membrane IL-6Rα, as well as IL-6 trans-signaling through soluble IL-6Rα, on the outcome of malaria has remained
Martin Whitham et al.
The Journal of biological chemistry, 287(14), 10771-10779 (2012-02-22)
Exercise increases the expression of the prototypical myokine IL-6, but the precise mechanism by which this occurs has yet to be identified. To mimic exercise conditions, C2C12 myotubes were mechanically stimulated via electrical pulse stimulation (EPS). We compared the responses
Shami Kanekar et al.
Journal of neuroscience research, 87(6), 1424-1434 (2008-12-31)
One premise regarding the mechanism of injury-evoked neuroregeneration is that injured cells induce the release of neurotrophic factors to trigger neurogenesis. Extracellular purine nucleotides exert multiple neurotrophic actions in the central nervous system mediated via activation of purinergic receptors. However

Nuestro equipo de científicos tiene experiencia en todas las áreas de investigación: Ciencias de la vida, Ciencia de los materiales, Síntesis química, Cromatografía, Analítica y muchas otras.

Póngase en contacto con el Servicio técnico