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Documenti fondamentali

EHU052811

Sigma-Aldrich

MISSION® esiRNA

targeting human BMPER

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About This Item

Codice UNSPSC:
41105324
NACRES:
NA.51

Descrizione

Powered by Eupheria Biotech

Livello qualitativo

Nome Commerciale

MISSION®

Stato

lyophilized powder

Sequenza bersaglio del cDNA di esiRNA

CCTGGATGGCTACCTCTTGAAAGTGACCACCAAAGCAGGTTTGGAAATATCTTGGGATGGAGACAGTTTTGTAGAAGTCATGGCTGCGCCGCATCTCAAGGGCAAGCTCTGTGGTCTTTGTGGCAACTACAATGGACATAAACGTGATGACTTAATTGGTGGAGATGGAAACTTCAAGTTTGATGTGGATGACTTTGCTGAATCTTGGAGGGTGGAGTCCAATGAGTTCTGCAACAGACCTCAGAGAAAGCCAGTGCCTGAACTGTGTCAAGGGACAGTCAAGGTAAAGCTCCGGGCCCATCGAGAATGCCAAAAGCTCAAATCCTGGGAGTTTCAGACCTGCCACTCGACTGTGGACTACGCCACTTTCTACCGGTCCTGTGTGACAGACATGTGTGAATGTCCAGTCCATAAAAACTGTTATTGCGAGTCATTTTTGGCATATACCCGG

N° accesso Ensembl | uomo

N° accesso NCBI

Condizioni di spedizione

ambient

Temperatura di conservazione

−20°C

Informazioni sul gene

Descrizione generale

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Note legali

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Codice della classe di stoccaggio

10 - Combustible liquids

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable


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Yong Xi et al.
BioMed research international, 2020, 3607436-3607436 (2020-04-21)
BMPER has been reported to be associated with the biological behavior of a few malignant tumors, but the mechanism is still unclear. We aimed to detect BMPER expression in ovarian epithelial tumor tissues and its effects on their biological behaviors
Thomas Helbing et al.
Inflammation, 40(2), 442-453 (2016-12-21)
The endothelium serves as a selective barrier and controls the exchange of nutrients, hormones, and leukocytes between blood and tissues. Molecular mechanisms contributing to the pathogenesis of endothelial barrier dysfunction remain incompletely understood. Accumulating evidence implicates bone morphogenetic protein (BMP)-modulator

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