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GF01141333

Ruthenium

Ruthenium, microfoil, disks, 10mm, thinness 0.1μm, specific density 122μg/cm2, permanent mylar 3.5μm support, 99.9%

Sinonimo/i:

Ruthenium, RU004500

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About This Item

Formula empirica (notazione di Hill):
Ru
Numero CAS:
Peso molecolare:
101.07
Numero MDL:
Codice UNSPSC:
12141739
ID PubChem:
NACRES:
NA.23

Saggio

≥99.9%

Forma fisica

foil

Produttore/marchio commerciale

Goodfellow 011-413-33

Resistività

7.1 μΩ-cm, 0°C

diam. × spessore

10 mm × 0.1 μm

P. eboll.

3900 °C (lit.)

Punto di fusione

2310 °C (lit.)

Densità

12.45 g/cm3 (lit.)

Stringa SMILE

[Ru]

InChI

1S/Ru
KJTLSVCANCCWHF-UHFFFAOYSA-N

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Descrizione generale

For updated SDS information please visit www.goodfellow.com.

Note legali

Product of Goodfellow

Codice della classe di stoccaggio

13 - Non Combustible Solids

Classe di pericolosità dell'acqua (WGK)

nwg

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable


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Gregory S Smith et al.
Dalton transactions (Cambridge, England : 2003), 40(41), 10793-10800 (2011-08-23)
The introduction of multifunctionalities for tumour targeting is becoming a popular strategy toward the development of new therapeutic agents. In particular, the multifaceted potential of ruthenium(II)-arene complexes show great promise as chemotherapeutics. An ever-increasing number of papers dealing with the
Lutz Ackermann et al.
Topics in current chemistry, 292, 211-229 (2010-01-01)
Stoichiometric cycloruthenation reactions of substrates containing Lewis-basic functionalities set the stage for efficient ruthenium-catalyzed C-H bond functionalization reactions. Thereby, selective addition reactions of C-H bonds across alkenes or alkynes enabled atom-economical synthesis of substituted arenes. More recently, ruthenium-catalyzed direct arylation
Nicole L Fry et al.
Accounts of chemical research, 44(4), 289-298 (2011-03-03)
Nitric oxide (NO) can induce apoptosis (programmed cell death) at micromolar or higher doses. Although cell death via NO-induced apoptosis has been studied quite extensively, the targeted delivery of such doses of NO to infected or malignant tissues has not
E Tfouni et al.
Current medicinal chemistry, 17(31), 3643-3657 (2010-09-18)
The discovery of the involvement of nitric oxide (NO) in several physiological and pathophysiological processes launched a spectacular increase in studies in areas such as chemistry, biochemistry, and pharmacology. As a consequence, the development of NO donors or scavengers for
Aviva Levina et al.
Metallomics : integrated biometal science, 1(6), 458-470 (2009-11-01)
Interest in Ru anticancer drugs has been growing rapidly since NAMI-A ((ImH(+))[Ru(III)Cl(4)(Im)(S-dmso)], where Im = imidazole and S-dmso = S-bound dimethylsulfoxide) or KP1019 ((IndH(+))[Ru(III)Cl(4)(Ind)(2)], where Ind = indazole) have successfully completed phase I clinical trials and an array of other

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