GF87620328
Iron
rod, 500mm, diameter 30mm, as drawn, 98+%
Synonyme(s) :
Iron, FE007975
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About This Item
Produits recommandés
Essai
≥98%
Forme
rod
Fabricant/nom de marque
Goodfellow 876-203-28
Résistivité
9.71 μΩ-cm
pb
2750 °C (lit.)
Pf
1535 °C (lit.)
Densité
7.86 g/mL at 25 °C (lit.)
Chaîne SMILES
[Fe]
InChI
1S/Fe
Clé InChI
XEEYBQQBJWHFJM-UHFFFAOYSA-N
Description générale
For updated SDS information please visit www.goodfellow.com.
Informations légales
Product of Goodfellow
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Infection and immunity, 82(6), 2448-2459 (2014-04-02)
Staphylococcus lugdunensis is a coagulase-negative staphylococcus that is a commensal of humans and an opportunistic pathogen. It can cause a spectrum of infections, including those that are associated with the ability to form biofilm, such as occurs with endocarditis or
Biochemistry, 53(14), 2286-2294 (2014-03-22)
The Isd (iron-regulated surface determinant) system is a multiprotein transporter that allows bacterium Staphylococcus aureus to take up iron from hemoglobin (Hb) during human infection. In this system, IsdB is a cell wall-anchored surface protein that contains two near iron
Clinical nephrology, 81(4), 251-258 (2014-03-25)
Hyperphosphatemia in advanced chronic kidney disease (CKD) necessitates the use of phosphate binders. This in vitro study assessed phosphate binding and Fe release properties of the novel iron-based phosphate binder PA21. Phosphate adsorption and Fe release were assessed under conditions
Environmental and molecular mutagenesis, 55(3), 266-277 (2014-04-12)
DNA methylation is an epigenetic mechanism that drives phenotype and that can be altered by environmental exposures including radiation. The majority of human radiation exposures occur in a relatively low dose range; however, the biological response to low dose radiation
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our
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