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565784

3-Bromo-5-fluoro-2-propoxyphenylboronic acid

Name: 3-Bromo-5-fluoro-2-propoxyphenylboronic acid
Brand: ALDRICH

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About This Item

Formule empirique (notation de Hill):

C₉H₁₁BBrFO₃

Numéro CAS:

868272-84-8

Poids moléculaire :

276.90

Numéro MDL:

MFCD07369707

Code UNSPSC :

12352103

ID de substance PubChem :

24880256

Forme

solid

Pf

51-56 °C (lit.)

Chaîne SMILES

CCCOc1c(Br)cc(F)cc1B(O)O

InChI

1S/C9H11BBrFO3/c1-2-3-15-9-7(10(13)14)4-6(12)5-8(9)11/h4-5,13-14H,2-3H2,1H3

Clé InChI

XOBLNJQYFCQQTQ-UHFFFAOYSA-N

Autres remarques

Contains varying amounts of anhydride

Code de la classe de stockage

13 - Non Combustible Solids

Classe de danger pour l'eau (WGK)

WGK 3

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable

Équipement de protection individuelle

Eyeshields, Gloves, type N95 (US)

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Identification of a Notch transcriptomic signature for breast cancer.

Eike-Benjamin Braune et al.

Breast cancer research : BCR, 26(1), 4-4 (2024-01-04)

Dysregulated Notch signalling contributes to breast cancer development and progression, but validated tools to measure the level of Notch signalling in breast cancer subtypes and in response to systemic therapy are largely lacking. A transcriptomic signature of Notch signalling would

Carbon Ion Irradiation Downregulates Notch Signaling in Glioma Cell Lines, Impacting Cell Migration and Spheroid Formation.

Vivek Kumar et al.

Cells, 11(21) (2022-11-12)

Photon-based radiotherapy upregulates Notch signaling in cancer, leading to the acquisition of the stem cell phenotype and induction of invasion/migration, which contributes to the development of resistance to therapy. However, the effect of carbon ion radiotherapy (CIRT) on Notch signaling

Class II phosphatidylinositol 3-kinase-C2α is essential for Notch signaling by regulating the endocytosis of γ-secretase in endothelial cells.

Shota Shimizu et al.

Scientific reports, 11(1), 5199-5199 (2021-03-06)

The class II α-isoform of phosphatidylinositol 3-kinase (PI3K-C2α) plays a crucial role in angiogenesis at least in part through participating in endocytosis and, thereby, endosomal signaling of several cell surface receptors including VEGF receptor-2 and TGFβ receptor in vascular endothelial

In vitro atlas of dorsal spinal interneurons reveals Wnt signaling as a critical regulator of progenitor expansion.

Sandeep Gupta et al.

Cell reports, 40(3), 111119-111119 (2022-07-21)

Restoring sensation after injury or disease requires a reproducible method for generating large quantities of bona fide somatosensory interneurons. Toward this goal, we assess the mechanisms by which dorsal spinal interneurons (dIs; dI1-dI6) can be derived from mouse embryonic stem

Notch signaling is activated in knee-innervating dorsal root ganglia in experimental models of osteoarthritis joint pain.

Lai Wang et al.

Arthritis research & therapy, 25(1), 63-63 (2023-04-16)

We aimed to explore activation of the Notch signaling pathway in knee-innervating lumbar dorsal root ganglia (DRG) in the course of experimental osteoarthritis (OA) in mice, and its role in knee hyperalgesia. Cultured DRG cells were stimulated with the TLR4

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