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334677

Sigma-Aldrich

2-Imino-4-thiobiuret

99%

Synonym(s):

Amidinothiourea

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25 G
CHF 48.20

CHF 48.20


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25 G
CHF 48.20

About This Item

Linear Formula:
H2NC(=NH)NHCSNH2
CAS Number:
Molecular Weight:
118.16
EC Number:
MDL number:
UNSPSC Code:
12352100
PubChem Substance ID:
NACRES:
NA.22

CHF 48.20


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Assay

99%

mp

171-173 °C (lit.)

functional group

amine
thiourea

SMILES string

NC(=N)NC(N)=S

InChI

1S/C2H6N4S/c3-1(4)6-2(5)7/h(H6,3,4,5,6,7)

InChI key

OKGXJRGLYVRVNE-UHFFFAOYSA-N

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Signal Word

Warning

Hazard Statements

Hazard Classifications

Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3

Target Organs

Respiratory system

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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I N Ianvareva et al.
Farmakologiia i toksikologiia, 51(2), 47-50 (1988-03-01)
Redox-reactions of flavine (FN) and pyridine (PN) nucleotides of the cell to guthimine were studied by the method of fluorescent microscopy without using any stains. The comparison was made of the kinetics of fluorescence intensity of FN and PN of
A A Waĭnson et al.
Radiobiologiia, 26(3), 329-333 (1986-05-01)
It is shown that such substances as gutimine, antizol and mexamine increases the resistance of animals to short-term breathing of gas mixtures containing 6 and 5% oxygen. Even if some of them decrease the degree of radioprotective effect of hypoxia
[Effect of antioxidants of the gutimine group on the course and outcome of the post-resuscitation process].
I E Trubina et al.
Patologicheskaia fiziologiia i eksperimental'naia terapiia, (5)(5), 12-15 (1986-09-01)
C Vértesi
Medical hypotheses, 40(6), 335-341 (1993-06-01)
S-allylgutimine has immunostimulant and tumor cell inhibitory activities. This complex action may be a result of the stimulation of IL-2 production. The effectiveness of immunotherapy depends on the uncoupling of IL-2 downregulation. This principle can be widely used with those
V T Dolgikh
Voprosy meditsinskoi khimii, 34(1), 33-38 (1988-01-01)
Long-term impairments of energy metabolisms in myocardium were found in rats after clinical death caused by acute hemorrhage. Bioenergetics of myocardium tended to normalize within a month after resuscitation. Severity of postresuscitation period correlated with the level of deteriorations observed

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