06720
2-Aminoethyl hydrogen sulfate
≥98.0% (T)
Synonym(s):
Sulfuric acid mono 2-aminoethylester
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About This Item
Linear Formula:
NH2CH2CH2OSO3H
CAS Number:
Molecular Weight:
141.15
Beilstein:
1704079
EC Number:
MDL number:
UNSPSC Code:
12352100
PubChem Substance ID:
NACRES:
NA.22
Assay:
≥98.0% (T)
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Quality Level
Assay
≥98.0% (T)
mp
277 °C (dec.) (lit.)
functional group
amine
SMILES string
NCCOS(O)(=O)=O
NCCOS(O)(=O)=O
InChI
1S/C2H7NO4S/c3-1-2-7-8(4,5)6/h1-3H2,(H,4,5,6)
InChI key
WSYUEVRAMDSJKL-UHFFFAOYSA-N
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Related Categories
Signal Word
Warning
Hazard Statements
Precautionary Statements
Hazard Classifications
Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
Target Organs
Respiratory system
Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Personal Protective Equipment
dust mask type N95 (US), Eyeshields, Gloves
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A E Herbison et al.
Journal of neurochemistry, 55(5), 1617-1623 (1990-11-01)
The characteristics of gamma-aminobutyric acid (GABA) release as monitored by microdialysis have been investigated in the chloral hydrate anaesthetised rat. The high outflow of GABA following insertion of the microdialysis probe (membrane 2 mm in length, 0.5 mm in diameter)
J P Loeffler et al.
Neuroendocrinology, 43(4), 504-510 (1986-01-01)
The inhibitory action of gamma-aminobutyric acid (GABA) on prolactin (PRL) messenger ribonucleic acid (mRNA) levels was studied in vitro in rat anterior pituitary cells in culture and in intact rats in vivo. PRL mRNA levels were determined by hybridization of
R Horton et al.
General pharmacology, 19(3), 403-405 (1988-01-01)
1. Oral administration of the GABA transaminase inhibitor ethanolamine-O-sulphate (EOS, 5 mg/ml in drinking water) to rats for 14 days suppressed food intake by 24%, but reduced weight gain by over 35%. 2. Thus, feed efficiency (g gain/MJ eaten) was
M Qume et al.
Biochemical pharmacology, 52(9), 1355-1363 (1996-11-08)
The inhibitory neurotransmitter gamma-aminobutyric acid (GABA) is not solely located in the CNS, it and the enzymes responsible for its synthesis (glutamic acid decarboxylase, GAD, EC 4.1.1.15) and catabolism (GABA-transaminase, GABA-T, EC 2.6.1.19) are also present in non-neuronal organs. Following
J Semba et al.
Neuropsychobiology, 21(3), 152-156 (1989-01-01)
We carried out the forced swimming test in mice to investigate the antidepressant potentials of GABA transaminase (GABA-T) inhibitors including aminooxyacetic acid, ethanolamine-O-sulfate, gamma-vinyl GABA (GVG) and valproic acid (VPA). In acute experiments only GVG reduced immobility. Following chronic oral
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