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A6855

Sigma-Aldrich

Arachidonoyl-AMC

fatty acid amide hydrolase substrate, 99% (HPLC), solid

Synonym(s):

AAMCA

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About This Item

Empirical Formula (Hill Notation):
C30H39NO3
Molecular Weight:
461.64
MDL number:
MFCD08062144
UNSPSC Code:
12352204
PubChem Substance ID:
24891145
NACRES:
NA.32
Pricing and availability is not currently available.

Product Name

Arachidonoyl-AMC, fatty acid amide hydrolase substrate

Quality Level

100

Assay

99% (HPLC)

form

solid

storage condition

under inert gas

solubility

DMSO: soluble

storage temp.

−20°C

SMILES string

CCCCC\C=C/C\C=C/C\C=C/C\C=C/CCCC(=O)Nc1ccc2C(C)=CC(=O)Oc2c1

InChI

1S/C30H39NO3/c1-3-4-5-6-7-8-9-10-11-12-13-14-15-16-17-18-19-20-29(32)31-26-21-22-27-25(2)23-30(33)34-28(27)24-26/h7-8,10-11,13-14,16-17,21-24H,3-6,9,12,15,18-20H2,1-2H3,(H,31,32)/b8-7-,11-10-,14-13-,17-16-

InChI key

IQPBUDMTXHWSHB-ZKWNWVNESA-N

Application

Fluorogenic substrate used in a selective and sensitive assay of fatty acid amide hydrolase (FAAH).

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

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Certificates of Analysis (COA)

Lot/Batch Number
MKCV0794
MKCS7574
MKCM6219
MKCB4120
MKBZ8452V

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Simona Di Martino et al.
Journal of medicinal chemistry, 63(7), 3634-3664 (2020-03-17)
Sphingolipids (SphLs) are a diverse class of molecules that are regulated by a complex network of enzymatic pathways. A disturbance in these pathways leads to lipid accumulation and initiation of several SphL-related disorders. Acid ceramidase is one of the key
Enrico Dainese et al.
Scientific reports, 10(1), 2292-2292 (2020-02-12)
Fatty acid amide hydrolase (FAAH) is a membrane-bound homodimeric enzyme that in vivo controls content and biological activity of N-arachidonoylethanolamine (AEA) and other relevant bioactive lipids termed endocannabinoids. Parallel orientation of FAAH monomers likely allows both subunits to simultaneously recruit
Xinwen Zhang et al.
Neuropharmacology, 128, 269-281 (2017-10-25)
Monoacylglycerol lipase (MGL) hydrolyzes 2-arachidonoylglycerol to arachidonic acid and glycerol. Inhibition of MGL may attenuate neuroinflammation by enhancing endocannabinoid signaling and decreasing prostaglandin (PG) production. Almost half of HIV infected individuals are afflicted with HIV-associated neurocognitive disorder (HAND), a neuroinflammatory

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