A8784
N-Acetylputrescine hydrochloride
≥98% (TLC)
Synonym(s):
Monoacetyl-1,4-diaminobutane hydrochloride, N-(4-Aminobutyl)acetamide hydrochloride
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About This Item
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Quality Level
Assay
≥98% (TLC)
form
solid
storage temp.
2-8°C
SMILES string
Cl.CC(=O)NCCCCN
InChI
1S/C6H14N2O.ClH/c1-6(9)8-5-3-2-4-7;/h2-5,7H2,1H3,(H,8,9);1H
InChI key
XBECFEJUQZXMFE-UHFFFAOYSA-N
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Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Personal Protective Equipment
dust mask type N95 (US), Eyeshields, Gloves
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Journal of chromatography, 233, 29-38 (1982-12-10)
A gas chromatographic method was developed for the determination of monoacetylputrescine, monoacetylcadaverine, N1-acetylspermidine and N8-acetylspermidine in human urine. The amines were isolated from urine by silica gel column chromatography. 1,10-Diaminodecane was used as internal standard. The amines were reacted with
Journal of immunology (Baltimore, Md. : 1950), 135(6), 3772-3776 (1985-12-01)
N,N'-Diacetylputrescine (tetramethylenebisacetamide [TMBA]) and its six carbon analog, hexamethylenebisacetamide (HMBA), inhibited the proliferative response of human B lymphocytes to anti-mu and formalinized Cowan I strain Staphylococcal aureus (SAC) stimulation. In contrast, B cell growth factor-stimulated proliferation of human B cells
Cancer research, 43(8), 3944-3947 (1983-08-01)
Polyamines are small, highly charged, organic cations of possible regulatory importance in RNA-dependent protein synthesis, the production of which reflects cellular growth and division. The cytokinetics of normal cell populations is circadian rhythmic. This is reflected by a circadian rhythmicity
Microbios, 73(294), 7-21 (1993-01-01)
1,3-Diaminopropane has been identified as the major polyamine of Acanthamoeba culbertsoni. N-acetylputrescine and spermidine were present in appreciable amounts and putrescine as well as N-acetylspermidine were also detected, but spermine was absent. Changes in polyamine levels were observed during the
Journal of biochemical toxicology, 5(1), 23-32 (1990-01-01)
The mechanism by which chlordecone (CD) amplifies the hepatotoxicity of halomethanes such as CCl4, CHCl3, and BrCCl3 has been a subject of intense study. Recent work has shown that suppression of hepatocellular regeneration leads to accelerated progression of liver injury
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