MAB303
Anti-Acetylcholinesterase Antibody, clone AE-1
clone AE-1, Chemicon®, from mouse
Synonym(s):
Anti-ACEE, Anti-N-ACHE, Anti-YT
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About This Item
UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41
Recommended Products
biological source
mouse
Quality Level
antibody form
purified antibody
antibody product type
primary antibodies
clone
AE-1, monoclonal
species reactivity
canine, bovine, human, chicken, guinea pig, monkey
manufacturer/tradename
Chemicon®
technique(s)
ELISA: suitable
immunohistochemistry: suitable
immunoprecipitation (IP): suitable
isotype
IgG1
NCBI accession no.
UniProt accession no.
shipped in
wet ice
target post-translational modification
unmodified
Gene Information
human ... ACHE(43)
Specificity
Acetylcholinesterase from human and monkey.
Immunogen
Acetylcholinesterase AChE from human erythrocyte.
Application
ELISA
Immunohistochemistry
Western Blot
Optimal working dilutions must be determined by end user.
Immunohistochemistry
Western Blot
Optimal working dilutions must be determined by end user.
Research Category
Neuroscience
Neuroscience
Research Sub Category
Neurotransmitters & Receptors
Neuronal & Glial Markers
Neurotransmitters & Receptors
Neuronal & Glial Markers
This Anti-Acetylcholinesterase Antibody, clone AE-1 is validated for use in ELISA, IH, IP for the detection of Acetylcholinesterase.
Physical form
Format: Purified
Liquid in 0.02M PB, 0.25M NaCl, pH 7.6, with 0.1% sodium azide.
Storage and Stability
Maintain at 2-8°C for up to 6 months.
Other Notes
Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Storage Class Code
10 - Combustible liquids
WGK
WGK 2
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
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Alicia J Dafferner et al.
Chemical research in toxicology, 30(10), 1897-1910 (2017-09-12)
Nerve agents and organophosphorus pesticides make a covalent bond with the active site serine of acetylcholinesterase (AChE), resulting in inhibition of AChE activity and toxic symptoms. AChE in red blood cells (RBCs) serves as a surrogate for AChE in the
Franziska Richter et al.
EBioMedicine, 41, 649-658 (2019-03-02)
The most prevalent inherited form of generalized dystonia is caused by a mutation in torsinA (DYT1, ∆GAG) with incomplete penetrance. Rodent models with mutated torsinA do not develop dystonic symptoms, but previous ex vivo studies indicated abnormal excitation of cholinergic
Pamela Jarrett et al.
The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques, 45(2), 150-157 (2018-03-07)
Neuronal ceroid-lipofuscinoses are a heterogeneous group of inherited disorders in which abnormal lipopigments form lysosomal inclusion bodies in neurons. Kufs disease is rare, and clinical symptoms include seizures, progressive cognitive impairment, and myoclonus. Most cases of Kufs disease are autosomal
Pia Pužar Dominkuš et al.
Journal of neurovirology, 23(5), 713-724 (2017-08-02)
HIV-1 infection of the central nervous system causes HIV-associated neurocognitive disorders, even in aviremic patients. Although astrocyte malfunction was associated to these disorders, their implication is overshadowed by contributions of microglia and macrophages. Astrocytes are infected with HIV-1 in vivo
Axodendritic contacts onto calcium/calmodulin-dependent protein kinase type II-expressing neurons in the barn owl auditory space map.
Rodriguez-Contreras, A; Liu, XB; DeBello, WM
The Journal of Neuroscience null
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