M4154
β-N-Methylamino-L-alanine hydrochloride
Sinónimos:
L-BMAA
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About This Item
Fórmula empírica (notación de Hill):
C4H10N2O2 · HCl
Número de CAS:
Peso molecular:
154.60
Número MDL:
Código UNSPSC:
12352200
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temp. de almacenamiento
2-8°C
cadena SMILES
CNCC(N)C(O)=O
Acciones bioquímicas o fisiológicas
Neuroexcitotoxic amino acid; may be involved in Guam amyotrophic lateral sclerosis
Código de clase de almacenamiento
13 - Non Combustible Solids
Clase de riesgo para el agua (WGK)
WGK 3
Punto de inflamabilidad (°F)
Not applicable
Punto de inflamabilidad (°C)
Not applicable
Equipo de protección personal
Eyeshields, Gloves, type N95 (US)
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András Guseo
Orvosi hetilap, 153(52), 2060-2069 (2012-12-25)
Parkinson's disease is one of the most frequent progressive degenerative disorders with unknown origin of the nervous system. The commutation of the disease on Guam led to the discovery of a neurotoxin which was also found in other continents. This
Elijah W Stommel et al.
Medical hypotheses, 80(2), 142-145 (2012-12-19)
Sporadic amyotrophic lateral sclerosis (sALS) is a fatal neurodegenerative disease with no known cause. There are many clues to suggest an environmental trigger for the disease, including reports of conjugal couples and co-localized employees that developed sALS. On the island
Travis Rush et al.
Neuroreport, 23(4), 216-219 (2012-02-09)
Determination of the environmental factors involved in neurodegenerative diseases has been elusive. Methylmercury and β-N-methylamino-L-alanine (BMAA) have both been implicated in this role. Exposure of primary cortical cultures to these compounds independently induced concentration-dependent neurotoxicity. Importantly, concentrations of BMAA (10-100
Liying Jiang et al.
Analytical and bioanalytical chemistry, 405(4), 1283-1292 (2012-11-28)
The cyanobacterial neurotoxin β-N-methylamino-L-alanine (BMAA) is an amino acid that is putatively associated with the pathology of amyotrophic lateral sclerosis/Parkinsonism-dementia complex (ALS-PDC) disease. It raises serious health risk concerns since cyanobacteria are ubiquitous thus making human exposure almost inevitable. The
Oskar Karlsson et al.
Toxicological sciences : an official journal of the Society of Toxicology, 130(2), 391-404 (2012-08-09)
The cyanobacterial toxin β-N-methylamino-L-alanine (BMAA) has been proposed to contribute to neurodegenerative disease. We have previously reported a selective uptake of BMAA in the mouse neonatal hippocampus and that exposure during the neonatal period causes learning and memory impairments in
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