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Merck

764981

Sigma-Aldrich

9,10-Bis(2-naphthyl)anthrace

99% (HPLC)

Sinónimos:

9,10-Di-2-naphthalenyl-anthracene, ADN

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About This Item

Fórmula empírica (notación de Hill):
C34H22
Número de CAS:
Peso molecular:
430.54
MDL number:
UNSPSC Code:
12352103
PubChem Substance ID:
NACRES:
NA.23

assay

99% (HPLC)

form

solid

mp

383-387 °C

SMILES string

c1ccc2cc(ccc2c1)-c3c4ccccc4c(-c5ccc6ccccc6c5)c7ccccc37

InChI

1S/C34H22/c1-3-11-25-21-27(19-17-23(25)9-1)33-29-13-5-7-15-31(29)34(32-16-8-6-14-30(32)33)28-20-18-24-10-2-4-12-26(24)22-28/h1-22H

InChI key

VIZUPBYFLORCRA-UHFFFAOYSA-N

Application

Fluorescent host material in high efficiency pure blue devices.

pictograms

Exclamation mark

signalword

Warning

hcodes

Hazard Classifications

Acute Tox. 4 Oral

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable


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S100B protein, previously proposed as a consolidated marker of brain damage in congenital heart disease (CHD) newborns who underwent cardiac surgery and cardiopulmonary bypass (CPB), has been progressively abandoned due to S100B CNS extra-source such as adipose tissue. The present
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Revista espanola de cardiologia (English ed.), 72(4), 317-323 (2018-04-16)
Cell-free DNA (cfDNA) in ST-segment elevation myocardial infarction might originate from hyperactivated leukocytes at the coronary lesion. Our aim was to investigate the relationship between cfDNA and coronary reperfusion. We studied 116 patients treated with primary angioplasty using thrombus aspiration.
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Oncotarget, 6(42), 44108-44122 (2015-12-18)
Ketamine enhances autonomic activity, and unmyelinated C-type baroreceptor afferents are more susceptible to be blocked by ketamine than myelinated A-types. However, the presynaptic transmission block in low-threshold and sex-specific myelinated Ah-type baroreceptor neurons (BRNs) is not elucidated. Action potentials (APs)
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Extended-spectrum β-lactamase (ESBL), plasmid-mediated AmpC (pAmpC) and MCR-1 phosphoethanolamine transferase enzymes have been pointed out as the main plasmid-mediated mechanisms of resistance to third generation cephalosporins (TGC) and colistin, respectively, and are currently considered a major concern both in human

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