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Key Documents

20-157

Sigma-Aldrich

TE Buffer - for use in ChIP Assays

For use in Chromatin Immunoprecipitation assays.

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About This Item

UNSPSC Code:
41116012
eCl@ss:
32160405
NACRES:
NA.41

Quality Level

form

liquid

manufacturer/tradename

Upstate®

shipped in

wet ice

Related Categories

Application

For use in Chromatin Immunoprecipitation assays.
Research Category
All

Physical form

10mM Tris-HCl, pH 8.0, 1mM EDTA

Storage and Stability

1 year at 4°C

Legal Information

UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Storage Class Code

12 - Non Combustible Liquids

WGK

nwg

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


Certificates of Analysis (COA)

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Haiyan Lei et al.
Development (Cambridge, England), 136(8), 1241-1249 (2009-03-06)
Previous work in C. elegans has shown that posterior embryonic bodywall muscle lineages are regulated through a genetically defined transcriptional cascade that includes PAL-1/Caudal-mediated activation of muscle-specific transcription factors, including HLH-1/MRF and UNC-120/SRF, which together orchestrate specification and differentiation. Using
Liang Xu et al.
Cancer letters, 482, 74-89 (2020-04-20)
Distant metastasis is the major cause of short survival in ccRCC patients. However, the development of effective therapies for metastatic ccRCC is limited. Herein, we reported that ETV4 was selected from among 150 relevant genes with in vivo evidence of
Yu-Hui Li et al.
Frontiers in oncology, 10, 567-567 (2020-05-22)
Global incidence and mortality associated with hepatocellular carcinoma (HCC) is steadily increasing. Metastasis-associated 1 (MTA1) can induce tumorigenesis and metastatic progression in HCC. However, the mechanistic details of MTA1-mediated regulation of HCC has not been completely defined. Epigenetic histone modification

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