539844
Goat Anti-Rabbit γ-Globulin
lyophilized, Calbiochem®
Synonym(s):
GARGG
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125 U
$136.00
About This Item
UNSPSC Code:
12352203
NACRES:
NA.46
Recommended Products
biological source
goat
Quality Level
antibody form
serum
clone
polyclonal
form
lyophilized
does not contain
preservative
manufacturer/tradename
Calbiochem®
storage condition
OK to freeze
isotype
IgG
shipped in
ambient
storage temp.
2-8°C
target post-translational modification
unmodified
General description
Goat polyclonal antibody supplied as lyophilized, undiluted serum. Recognizes rabbit γ-globulin.
Note: one unit is equivalent to one RIA tube.
One unit of goat anti-rabbit γ-globulin (GARGG) contains enough antibody to maximally precipitate the γ-globulin in 200 µl of 2% normal rabbit serum.
This Goat Anti-Rabbit γ-Globulin is validated for use in Immunoelectrophoresis, Radioimmunoassay for the detection of Rabbit γ-Globulin.
Application
Immunoelectrophoresis (see comments)
Radioimmunoassay (see comments)
Radioimmunoassay (see comments)
Warning
Toxicity: Standard Handling (A)
Physical form
Undiluted serum.
Reconstitution
Reconstitute with 12.5 ml H₂O. Following reconstitution, aliquot and freeze (-20°C). Stock solutions are stable for up to 4 weeks at -20°C.
Other Notes
One unit of goat anti-rabbit γ-globulin (GARGG) contains enough antibody to maximally precipitate the γ-globulin in 200 µl of 2% normal rabbit serum. Characteristic precipitin line is observed with normal rabbit serum by immunoelectrophoresis. This antibody has also been reported to work for RIA. Variables associated with assay conditions will dictate the proper working dilution.
Legal Information
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
Storage Class
11 - Combustible Solids
wgk_germany
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Certificates of Analysis (COA)
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John D Johnson et al.
American journal of physiology. Regulatory, integrative and comparative physiology, 284(2), R422-R432 (2002-10-26)
It has recently been reported that exposure to inescapable tailshock (IS) enhances the release of proinflammatory cytokines following bacterial challenge. However, it is not known whether the level of potentiation of proinflammatory cytokines is sufficient to exaggerate any of the
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