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Merck
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Documentos

23-0850

Sigma-Aldrich

Oxalic acid solution

0.5 M

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About This Item

Fórmula lineal:
HOOCCOOH
Número de CAS:
144-62-7
Peso molecular:
90.03
MDL number:
MFCD00002573
UNSPSC Code:
12352106
PubChem Substance ID:
329752381

form

liquid

availability

available only in Japan

concentration

0.5 M
1 N

density

1.02 g/cm3 at 20 °C

SMILES string

OC(=O)C(O)=O

InChI

1S/C2H2O4/c3-1(4)2(5)6/h(H,3,4)(H,5,6)

InChI key

MUBZPKHOEPUJKR-UHFFFAOYSA-N

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pictograms

Corrosion
GHS05

signalword

Danger

hcodes

H318

Hazard Classifications

Eye Dam. 1

Storage Class

12 - Non Combustible Liquids

wgk_germany

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable

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L K Massey et al.
Journal of agricultural and food chemistry, 49(9), 4262-4266 (2001-09-18)
Consumption of soybeans and food products made from them is increasing because of their desirable nutritional value. However, the oxalate content of seeds from 11 cultivars of soybean showed relatively high levels of total oxalate from 0.67 to 3.5 g/100
Y Nakagawa et al.
The Journal of clinical investigation, 76(4), 1455-1462 (1985-10-01)
One reason that some people are prone to calcium oxalate nephrolithiasis is that they produce urine that is subnormal in its ability to inhibit the growth of calcium oxalate crystals. We have identified in human urine a glycoprotein (GCI) that
G M Gadd
Advances in microbial physiology, 41, 47-92 (1999-09-29)
The production of organic acids by fungi has profound implications for metal speciation, physiology and biogeochemical cycles. Biosynthesis of oxalic acid from glucose occurs by hydrolysis of oxaloacetate to oxalate and acetate catalysed by cytosolic oxaloacetase, whereas on citric acid
R de Water et al.
American journal of kidney diseases : the official journal of the National Kidney Foundation, 33(4), 761-771 (1999-04-09)
Urinary calcium oxalate (CaOx) crystals and crystal agglomerates are normally harmlessly excreted, but in nephrolithiasis they are retained by tubular epithelial cells and shifted into the renal interstitium. This crystalline material induces an inflammatory response consisting of an increase in
A Amoroso et al.
Journal of the American Society of Nephrology : JASN, 12(10), 2072-2079 (2001-09-20)
Primary hyperoxaluria type 1 (PH1) is an autosomal recessive disorder that is caused by a deficiency of alanine: glyoxylate aminotransferase (AGT), which is encoded by a single copy gene (AGXT). Molecular diagnosis was used in conjunction with clinical, biochemical, and
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