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Key Documents

AP147F

Sigma-Aldrich

Rabbit Anti-Sheep IgG Antibody, FITC conjugate

Chemicon®, from rabbit

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.46

biological source

rabbit

Quality Level

conjugate

FITC conjugate

antibody form

affinity purified immunoglobulin

antibody product type

secondary antibodies

clone

polyclonal

species reactivity

sheep

manufacturer/tradename

Chemicon®

technique(s)

immunofluorescence: suitable

shipped in

wet ice

target post-translational modification

unmodified

Categorías relacionadas

Specificity

Sheep IgG (H&L)

Immunogen

Purified Sheep IgG.

Application

DILUTION:

Optimal working dilution must be determined by the end user.
Rabbit anti-Sheep IgG Antibody, FITC conjugate is an antibody against Sheep IgG for use in IF.
Research Category
Secondary & Control Antibodies
Research Sub Category
Whole Immunoglobulin Secondary Antibodies

Physical form

Lyophilized from 0.02M Phosphate buffer, 0.25 M NaCl with 15 mg/mL BSA and 0.1% sodium azide.

RECONSTITUTION:

Reconstitute to 1 mg/mL with sterile distilled water.

Storage and Stability

Maintain lyophilized material at 2-8°C up to one year. After reconstitution maintain at 2-8°C in an undiluted format for up to 6 months.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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hcodes

Hazard Classifications

Aquatic Chronic 3

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3


Certificados de análisis (COA)

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Visite la Librería de documentos

C Giaroni et al.
Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society, 25(2), e114-e126 (2013-01-03)
Intestinal ischemia and reperfusion (I/R) injury leads to abnormalities in motility, namely delay of transit, caused by damage to myenteric neurons. Alterations of the nitrergic transmission may occur in these conditions. This study investigated whether an in vitro I/R injury

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