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05-782

Sigma-Aldrich

Anti-GST Tag Antibody

Upstate®, from mouse

Synonym(s):

Anti-DFN7, Anti-FAEES3, Anti-GST3, Anti-GSTP, Anti-HEL-S-22, Anti-PI

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.43

biological source

mouse

Quality Level

antibody form

purified antibody

antibody product type

primary antibodies

clone

monoclonal

species reactivity

bacteria

manufacturer/tradename

Upstate®

technique(s)

western blot: suitable

isotype

IgG

NCBI accession no.

UniProt accession no.

shipped in

dry ice

target post-translational modification

unmodified

Specificity

GST fusion proteins
Not expected to cross-react with GST from other species.

Immunogen

Recombinant GST (glutathione S-transferase) from Shistosoma japonicum expressed in E. coli

Application

Detect GST Tag using this Anti-GST Tag Antibody validated for use in WB.

Quality

routinely evaluated by immunoblot on recombinant GST protein (Catalog #12-350) and GST-Abltide fusion protein (Catalog #12-525)

Target description

varies depending upon the GST fusion proteins being recognized

Physical form

Format: Purified
protein G purified mouse IgG in storage buffer (PBS containing 0.05% sodium azide) and 30% glycerol

Legal Information

UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Polarity underlies all directional growth responses in plants including growth towards the light (phototropism). The plasma-membrane associated protein, NON-PHOTOTROPIC HYPOCOTYL 3 (NPH3) is a key determinant of phototropic growth which is regulated by phototropin (phot) AGC kinases. Here we demonstrate
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Ataxia Telangiectasia mutated and RAD3-related (ATR) kinase is activated by DNA replication stress and also by various forms of DNA damage, including DNA double-strand breaks (DSBs). Recruitment to sites of damage is insufficient for ATR activation as one of two

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